勝木 陽子

Last Update: 2021/06/28 18:11:21

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Name(Kanji/Kana/Abecedarium Latinum)
勝木 陽子/カツキ ヨウコ/Katsuki, Yoko
Primary Affiliation(Org1/Job title)
Graduate School of Biostudies/Program-Specific Senior Lecturer/Program-Specific Junior Associate Professor
Academic Degree
Field(Japanese) Field(English) University(Japanese) University(English) Method
博士(医学) 東京医科歯科大学
Language of Instruction
Language(japanese) Language(english) Code
日本語 Japanese jpn
researchmap URL
https://researchmap.jp/read0123721
Published Papers
Author Author(Japanese) Author(English) Title Title(Japanese) Title(English) Bibliography Bibliography(Japanese) Bibliography(English) Publication date Refereed paper Language Publishing type Disclose
Yusuke Okamoto, Masako Abe, Anfeng Mu, Yasuko Tempaku, Colette B Rogers, Ayako L Mochizuki, Yoko Katsuki, Masato T Kanemaki, Akifumi Takaori-Kondo, Alexandra T Sobeck, Anja-Katrin Bielinsky, Minoru Takata Yusuke Okamoto, Masako Abe, Anfeng Mu, Yasuko Tempaku, Colette B Rogers, Ayako L Mochizuki, Yoko Katsuki, Masato T Kanemaki, Akifumi Takaori-Kondo, Alexandra T Sobeck, Anja-Katrin Bielinsky, Minoru Takata Yusuke Okamoto, Masako Abe, Anfeng Mu, Yasuko Tempaku, Colette B Rogers, Ayako L Mochizuki, Yoko Katsuki, Masato T Kanemaki, Akifumi Takaori-Kondo, Alexandra T Sobeck, Anja-Katrin Bielinsky, Minoru Takata SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells. SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells. SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells. Blood Blood Blood 2020/07/31 Refereed English Research paper(scientific journal) Disclose to all
Yoko Katsuki, Penny A. Jeggo, Yuki Uchihara, Minoru Takata, Atsushi Shibata Yoko Katsuki, Penny A. Jeggo, Yuki Uchihara, Minoru Takata, Atsushi Shibata Yoko Katsuki, Penny A. Jeggo, Yuki Uchihara, Minoru Takata, Atsushi Shibata DNA double-strand break end resection: a critical relay point for determining the pathway of repair and signaling DNA double-strand break end resection: a critical relay point for determining the pathway of repair and signaling DNA double-strand break end resection: a critical relay point for determining the pathway of repair and signaling Genome Instability & Disease, 1, 4, 155-171 Genome Instability & Disease, 1, 4, 155-171 Genome Instability & Disease, 1, 4, 155-171 2020/07 Refereed Research paper(scientific journal) Disclose to all
Shojiro Inano, Koichi Sato, Yoko Katsuki, Wataru Kobayashi, Hiroki Tanaka, Kazuhiro Nakajima, Shinichiro Nakada, Hiroyuki Miyoshi, Kerstin Knies, Akifumi Takaori-Kondo, Detlev Schindler, Masamichi Ishiai, Hitoshi Kurumizaka, Minoru Takata Shojiro Inano, Koichi Sato, Yoko Katsuki, Wataru Kobayashi, Hiroki Tanaka, Kazuhiro Nakajima, Shinichiro Nakada, Hiroyuki Miyoshi, Kerstin Knies, Akifumi Takaori-Kondo, Detlev Schindler, Masamichi Ishiai, Hitoshi Kurumizaka, Minoru Takata Shojiro Inano, Koichi Sato, Yoko Katsuki, Wataru Kobayashi, Hiroki Tanaka, Kazuhiro Nakajima, Shinichiro Nakada, Hiroyuki Miyoshi, Kerstin Knies, Akifumi Takaori-Kondo, Detlev Schindler, Masamichi Ishiai, Hitoshi Kurumizaka, Minoru Takata RFWD3-Mediated Ubiquitination Promotes Timely Removal of Both RPA and RAD51 from DNA Damage Sites to Facilitate Homologous Recombination RFWD3-Mediated Ubiquitination Promotes Timely Removal of Both RPA and RAD51 from DNA Damage Sites to Facilitate Homologous Recombination RFWD3-Mediated Ubiquitination Promotes Timely Removal of Both RPA and RAD51 from DNA Damage Sites to Facilitate Homologous Recombination MOLECULAR CELL, 66, 5, 622-+ MOLECULAR CELL, 66, 5, 622-+ MOLECULAR CELL, 66, 5, 622-+ 2017/06 Refereed English Research paper(scientific journal) Disclose to all
Koichi Sato, Mayo Shimomuki, Yoko Katsuki, Daisuke Takahashi, Wataru Kobayashi, Masamichi Ishiai, Hiroyuki Miyoshi, Minoru Takata, Hitoshi Kurumizaka Koichi Sato, Mayo Shimomuki, Yoko Katsuki, Daisuke Takahashi, Wataru Kobayashi, Masamichi Ishiai, Hiroyuki Miyoshi, Minoru Takata, Hitoshi Kurumizaka Koichi Sato, Mayo Shimomuki, Yoko Katsuki, Daisuke Takahashi, Wataru Kobayashi, Masamichi Ishiai, Hiroyuki Miyoshi, Minoru Takata, Hitoshi Kurumizaka FANCI-FANCD2 stabilizes the RAD51-DNA complex by binding RAD51 and protects the 5 '-DNA end FANCI-FANCD2 stabilizes the RAD51-DNA complex by binding RAD51 and protects the 5 '-DNA end FANCI-FANCD2 stabilizes the RAD51-DNA complex by binding RAD51 and protects the 5 '-DNA end NUCLEIC ACIDS RESEARCH, 44, 22, 10758-10771 NUCLEIC ACIDS RESEARCH, 44, 22, 10758-10771 NUCLEIC ACIDS RESEARCH, 44, 22, 10758-10771 2016/12 Refereed English Research paper(scientific journal) Disclose to all
Yoko Katsuki, Minoru Takata Yoko Katsuki, Minoru Takata Yoko Katsuki, Minoru Takata Defects in homologous recombination repair behind the human diseases: FA and HBOC Defects in homologous recombination repair behind the human diseases: FA and HBOC Defects in homologous recombination repair behind the human diseases: FA and HBOC ENDOCRINE-RELATED CANCER, 23, 10, T19-T37 ENDOCRINE-RELATED CANCER, 23, 10, T19-T37 ENDOCRINE-RELATED CANCER, 23, 10, T19-T37 2016/10 Refereed English Disclose to all
Meryem Alagoz, Yoko Katsuki, Hideaki Ogiwara, Tomoo Ogi, Atsushi Shibata, Andreas Kakarougkas, Penny Jeggo Meryem Alagoz, Yoko Katsuki, Hideaki Ogiwara, Tomoo Ogi, Atsushi Shibata, Andreas Kakarougkas, Penny Jeggo Meryem Alagoz, Yoko Katsuki, Hideaki Ogiwara, Tomoo Ogi, Atsushi Shibata, Andreas Kakarougkas, Penny Jeggo SETDB1, HP1 and SUV39 promote repositioning of 53BP1 to extend resection during homologous recombination in G2 cells SETDB1, HP1 and SUV39 promote repositioning of 53BP1 to extend resection during homologous recombination in G2 cells SETDB1, HP1 and SUV39 promote repositioning of 53BP1 to extend resection during homologous recombination in G2 cells NUCLEIC ACIDS RESEARCH, 43, 16, 7931-7944 NUCLEIC ACIDS RESEARCH, 43, 16, 7931-7944 NUCLEIC ACIDS RESEARCH, 43, 16, 7931-7944 2015/09 Refereed English Research paper(scientific journal) Disclose to all
Andreas Kakarougkas, Amani Ismail, Yoko Katsuki, Raimundo Freire, Atsushi Shibata, Penny A. Jeggo Andreas Kakarougkas, Amani Ismail, Yoko Katsuki, Raimundo Freire, Atsushi Shibata, Penny A. Jeggo Andreas Kakarougkas, Amani Ismail, Yoko Katsuki, Raimundo Freire, Atsushi Shibata, Penny A. Jeggo Co-operation of BRCA1 and POH1 relieves the barriers posed by 53BP1 and RAP80 to resection Co-operation of BRCA1 and POH1 relieves the barriers posed by 53BP1 and RAP80 to resection Co-operation of BRCA1 and POH1 relieves the barriers posed by 53BP1 and RAP80 to resection NUCLEIC ACIDS RESEARCH, 41, 22, 10298-10311 NUCLEIC ACIDS RESEARCH, 41, 22, 10298-10311 NUCLEIC ACIDS RESEARCH, 41, 22, 10298-10311 2013/12 Refereed English Research paper(scientific journal) Disclose to all
Yoko Katsuki, Shinichiro Nakada, Tetsuji Yokoyama, Issei Imoto, Johji Inazawa, Masayuki Nagasawa, Shuki Mizutani Yoko Katsuki, Shinichiro Nakada, Tetsuji Yokoyama, Issei Imoto, Johji Inazawa, Masayuki Nagasawa, Shuki Mizutani Yoko Katsuki, Shinichiro Nakada, Tetsuji Yokoyama, Issei Imoto, Johji Inazawa, Masayuki Nagasawa, Shuki Mizutani Caffeine yields aneuploidy through asymmetrical cell division caused by misalignment of chromosomes Caffeine yields aneuploidy through asymmetrical cell division caused by misalignment of chromosomes Caffeine yields aneuploidy through asymmetrical cell division caused by misalignment of chromosomes CANCER SCIENCE, 99, 8, 1539-1545 CANCER SCIENCE, 99, 8, 1539-1545 CANCER SCIENCE, 99, 8, 1539-1545 2008/08 Refereed English Research paper(scientific journal) Disclose to all
S Nakada, Y Katsuki, Imoto, I, T Yokoyama, M Nagasawa, J Inazawa, S Mizutani S Nakada, Y Katsuki, Imoto, I, T Yokoyama, M Nagasawa, J Inazawa, S Mizutani S Nakada, Y Katsuki, Imoto, I, T Yokoyama, M Nagasawa, J Inazawa, S Mizutani Early G2/M checkpoint failure as a molecular mechanism underlying etoposide-induced chromosomal aberrations Early G2/M checkpoint failure as a molecular mechanism underlying etoposide-induced chromosomal aberrations Early G2/M checkpoint failure as a molecular mechanism underlying etoposide-induced chromosomal aberrations JOURNAL OF CLINICAL INVESTIGATION, 116, 1, 80-89 JOURNAL OF CLINICAL INVESTIGATION, 116, 1, 80-89 JOURNAL OF CLINICAL INVESTIGATION, 116, 1, 80-89 2006/01 Refereed English Research paper(scientific journal) Disclose to all
R Tsuchida, T Yamada, M Takagi, A Shimada, C Ishioka, Y Katsuki, T Igarashi, L Chessa, D Delia, H Teraoka, S Mizutani R Tsuchida, T Yamada, M Takagi, A Shimada, C Ishioka, Y Katsuki, T Igarashi, L Chessa, D Delia, H Teraoka, S Mizutani R Tsuchida, T Yamada, M Takagi, A Shimada, C Ishioka, Y Katsuki, T Igarashi, L Chessa, D Delia, H Teraoka, S Mizutani Detection of ATM gene mutation in human glioma cell line M059J by a rapid frameshift/stop codon assay in yeast Detection of ATM gene mutation in human glioma cell line M059J by a rapid frameshift/stop codon assay in yeast Detection of ATM gene mutation in human glioma cell line M059J by a rapid frameshift/stop codon assay in yeast RADIATION RESEARCH, 158, 2, 195-201 RADIATION RESEARCH, 158, 2, 195-201 RADIATION RESEARCH, 158, 2, 195-201 2002/08 Refereed English Research paper(scientific journal) Disclose to all
Title language:
Misc
Author Author(Japanese) Author(English) Title Title(Japanese) Title(English) Bibliography Bibliography(Japanese) Bibliography(English) Publication date Refereed paper Language Publishing type Disclose
勝木 陽子, 高田 穣 勝木 陽子, 高田 穣 複製ストレス応答によるゲノム安定化メカニズム 複製ストレス応答によるゲノム安定化メカニズム 遺伝子医学, 30 遺伝子医学, 30 , 30 2019 Japanese Article, review, commentary, editorial, etc.(scientific journal) Disclose to all
Shinichiro Nakada, Yoko Katsuki, Issei Imoto, Tetsuji Yokoyama, Masayuki Nagasawa, Johji Inazawa, Shuki Mizutani Shinichiro Nakada, Yoko Katsuki, Issei Imoto, Tetsuji Yokoyama, Masayuki Nagasawa, Johji Inazawa, Shuki Mizutani Shinichiro Nakada, Yoko Katsuki, Issei Imoto, Tetsuji Yokoyama, Masayuki Nagasawa, Johji Inazawa, Shuki Mizutani Early G2/M checkpoint failure as a molecular mechanism underlying etoposide-induced chromosomal aberrations (vol 116, pg 80, 2006) Early G2/M checkpoint failure as a molecular mechanism underlying etoposide-induced chromosomal aberrations (vol 116, pg 80, 2006) Early G2/M checkpoint failure as a molecular mechanism underlying etoposide-induced chromosomal aberrations (vol 116, pg 80, 2006) JOURNAL OF CLINICAL INVESTIGATION, 116, 8, 2306-2307 JOURNAL OF CLINICAL INVESTIGATION, 116, 8, 2306-2307 JOURNAL OF CLINICAL INVESTIGATION, 116, 8, 2306-2307 2006/08 English Other Disclose to all
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Conference Activities & Talks
Title Title(Japanese) Title(English) Conference Conference(Japanese) Conference(English) Promotor Promotor(Japanese) Promotor(English) Date Language Assortment Disclose
Elucidation of the ubiquitination pathway mediating recruitment of SLX4 during ICL repair[Invited] ICL 修復因子 SLX4 のフォーカス形成に関与する 新規ユビキチン化経路の探索 [Invited] Elucidation of the ubiquitination pathway mediating recruitment of SLX4 during ICL repair [Invited] The 62nd Annual Meeting of the Japanese Radiation Research Society 日本放射線影響学会第62回大会 The 62nd Annual Meeting of the Japanese Radiation Research Society 2019/11/14 Japanese Symposium workshop panel(nominated) Disclose to all
DNAクロスリンク修復因子SLX4のユビキチン化による制御機構.[Invited] DNAクロスリンク修復因子SLX4のユビキチン化による制御機構. [Invited] 日本放射線影響学会第61回大会 日本放射線影響学会第61回大会 2018/11 Disclose to all
DNAクロスリンク切断酵素SLX4のユビキチン化による制御機構.[Invited] DNAクロスリンク切断酵素SLX4のユビキチン化による制御機構. [Invited] 第41回日本分子生物学会年会 第41回日本分子生物学会年会 2018/11 Disclose to all
DNAクロスリンク修復因子SLX4のユビキチン化による制御機構[Invited] DNAクロスリンク修復因子SLX4のユビキチン化による制御機構 [Invited] 日本遺伝学会第90回大会 日本遺伝学会第90回大会 2018/09 Disclose to all
ICL修復因子SLX4はRNF168依存的なユビキチン化経路を介して損傷部位に集積する.[Invited] ICL修復因子SLX4はRNF168依存的なユビキチン化経路を介して損傷部位に集積する. [Invited] 2017年度生命科学系学会合同年次大会 2017年度生命科学系学会合同年次大会 2017/12 Disclose to all
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External funds: competitive funds and Grants-in-Aid for Scientific Research (Kakenhi)
Type Position Title(Japanese) Title(English) Period
基盤研究(C) Representative BioIDシステムを利用したDNA修復因子SLX4の制御機構の解析 (2020年度分) 2020/04/01-2021/03/31