但馬 正樹

Last Update: 2021/06/29 14:34:02

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Name(Kanji/Kana/Abecedarium Latinum)
但馬 正樹/タジマ マサキ/Tajima, Masaki
Primary Affiliation(Org1/Job title)
Graduate School of Medicine/Assistant Professor
Faculty
Org1 Job title
医学部
Academic Degree
Field(Japanese) Field(English) University(Japanese) University(English) Method
修士(医科学) 北海道大学
博士(医学) 北海道大学
researchmap URL
https://researchmap.jp/masakitajima
Published Papers
Author Author(Japanese) Author(English) Title Title(Japanese) Title(English) Bibliography Bibliography(Japanese) Bibliography(English) Publication date Refereed paper Language Publishing type Disclose
Tomohiro Watanabe, Kosuke Minaga, Ken Kamata, Toshiharu Sakurai, Yoriaki Komeda, Tomoyuki Nagai, Atsushi Kitani, Masaki Tajima, Ivan J Fuss, Masatoshi Kudo, Warren Strober Tomohiro Watanabe, Kosuke Minaga, Ken Kamata, Toshiharu Sakurai, Yoriaki Komeda, Tomoyuki Nagai, Atsushi Kitani, Masaki Tajima, Ivan J Fuss, Masatoshi Kudo, Warren Strober Tomohiro Watanabe, Kosuke Minaga, Ken Kamata, Toshiharu Sakurai, Yoriaki Komeda, Tomoyuki Nagai, Atsushi Kitani, Masaki Tajima, Ivan J Fuss, Masatoshi Kudo, Warren Strober RICK/RIP2 is a NOD2-independent nodal point of gut inflammation. RICK/RIP2 is a NOD2-independent nodal point of gut inflammation. RICK/RIP2 is a NOD2-independent nodal point of gut inflammation. International immunology, 31, 10, 669-683 International immunology, 31, 10, 669-683 International immunology, 31, 10, 669-683 2019/09/18 English Research paper(scientific journal) Disclose to all
Tetsuya Takagawa, Atsushi Kitani, Ivan Fuss, Beth Levine, Steven R Brant, Inga Peter, Masaki Tajima, Shiro Nakamura, Warren Strober Tetsuya Takagawa, Atsushi Kitani, Ivan Fuss, Beth Levine, Steven R Brant, Inga Peter, Masaki Tajima, Shiro Nakamura, Warren Strober Tetsuya Takagawa, Atsushi Kitani, Ivan Fuss, Beth Levine, Steven R Brant, Inga Peter, Masaki Tajima, Shiro Nakamura, Warren Strober An increase in LRRK2 suppresses autophagy and enhances Dectin-1-induced immunity in a mouse model of colitis. An increase in LRRK2 suppresses autophagy and enhances Dectin-1-induced immunity in a mouse model of colitis. An increase in LRRK2 suppresses autophagy and enhances Dectin-1-induced immunity in a mouse model of colitis. Science translational medicine, 10, 444 Science translational medicine, 10, 444 Science translational medicine, 10, 444 2018/06/06 English Research paper(scientific journal) Disclose to all
Yasuyuki Arai, Uimook Choi, Cristina I Corsino, Sherry M Koontz, Masaki Tajima, Colin L Sweeney, Mary A Black, Steven A Feldman, Mary C Dinauer, Harry L Malech Yasuyuki Arai, Uimook Choi, Cristina I Corsino, Sherry M Koontz, Masaki Tajima, Colin L Sweeney, Mary A Black, Steven A Feldman, Mary C Dinauer, Harry L Malech Yasuyuki Arai, Uimook Choi, Cristina I Corsino, Sherry M Koontz, Masaki Tajima, Colin L Sweeney, Mary A Black, Steven A Feldman, Mary C Dinauer, Harry L Malech Myeloid Conditioning with c-kit-Targeted CAR-T Cells Enables Donor Stem Cell Engraftment. Myeloid Conditioning with c-kit-Targeted CAR-T Cells Enables Donor Stem Cell Engraftment. Myeloid Conditioning with c-kit-Targeted CAR-T Cells Enables Donor Stem Cell Engraftment. Molecular therapy : the journal of the American Society of Gene Therapy, 26, 5, 1181-1197 Molecular therapy : the journal of the American Society of Gene Therapy, 26, 5, 1181-1197 Molecular therapy : the journal of the American Society of Gene Therapy, 26, 5, 1181-1197 2018/05/02 English Research paper(scientific journal) Disclose to all
Takayuki Satoh, Masaki Tajima, Daiko Wakita, Hidemitsu Kitamura, Takashi Nishimura Takayuki Satoh, Masaki Tajima, Daiko Wakita, Hidemitsu Kitamura, Takashi Nishimura Takayuki Satoh, Masaki Tajima, Daiko Wakita, Hidemitsu Kitamura, Takashi Nishimura The development of IL-17/IFN-γ-double producing CTLs from Tc17 cells is driven by epigenetic suppression of Socs3 gene promoter. The development of IL-17/IFN-γ-double producing CTLs from Tc17 cells is driven by epigenetic suppression of Socs3 gene promoter. The development of IL-17/IFN-γ-double producing CTLs from Tc17 cells is driven by epigenetic suppression of Socs3 gene promoter. European journal of immunology, 42, 9, 2329-42 European journal of immunology, 42, 9, 2329-42 European journal of immunology, 42, 9, 2329-42 2012/09 English Research paper(scientific journal) Disclose to all
Masaki Tajima, Daiko Wakita, Takayuki Satoh, Hidemitsu Kitamura, Takashi Nishimura Masaki Tajima, Daiko Wakita, Takayuki Satoh, Hidemitsu Kitamura, Takashi Nishimura Masaki Tajima, Daiko Wakita, Takayuki Satoh, Hidemitsu Kitamura, Takashi Nishimura IL-17/IFN-γ double producing CD8+ T (Tc17/IFN-γ) cells: a novel cytotoxic T-cell subset converted from Tc17 cells by IL-12. IL-17/IFN-γ double producing CD8+ T (Tc17/IFN-γ) cells: a novel cytotoxic T-cell subset converted from Tc17 cells by IL-12. IL-17/IFN-γ double producing CD8+ T (Tc17/IFN-γ) cells: a novel cytotoxic T-cell subset converted from Tc17 cells by IL-12. International immunology, 23, 12, 751-9 International immunology, 23, 12, 751-9 International immunology, 23, 12, 751-9 2011/12 English Research paper(scientific journal) Disclose to all
Daisuke Noguchi, Daiko Wakita, Takayuki Ohkuri, Masaki Tajima, Kenji Chamoto, Hidemitsu Kitamura, Takashi Nishimura Daisuke Noguchi, Daiko Wakita, Takayuki Ohkuri, Masaki Tajima, Kenji Chamoto, Hidemitsu Kitamura, Takashi Nishimura Daisuke Noguchi, Daiko Wakita, Takayuki Ohkuri, Masaki Tajima, Kenji Chamoto, Hidemitsu Kitamura, Takashi Nishimura Blockade of IL-6-signaling inhibits the pathogenesis of CD4+ T cell-mediated lethal graft-versus-host reaction against minor histocompatibility antigen. Blockade of IL-6-signaling inhibits the pathogenesis of CD4+ T cell-mediated lethal graft-versus-host reaction against minor histocompatibility antigen. Blockade of IL-6-signaling inhibits the pathogenesis of CD4+ T cell-mediated lethal graft-versus-host reaction against minor histocompatibility antigen. Immunology letters, 136, 2, 146-55 Immunology letters, 136, 2, 146-55 Immunology letters, 136, 2, 146-55 2011/05 English Research paper(scientific journal) Disclose to all
Masaki Tajima, Daiko Wakita, Daisuke Noguchi, Kenji Chamoto, Zhang Yue, Kazunori Fugo, Harumichi Ishigame, Yoichiro Iwakura, Hidemitsu Kitamura, Takashi Nishimura Masaki Tajima, Daiko Wakita, Daisuke Noguchi, Kenji Chamoto, Zhang Yue, Kazunori Fugo, Harumichi Ishigame, Yoichiro Iwakura, Hidemitsu Kitamura, Takashi Nishimura Masaki Tajima, Daiko Wakita, Daisuke Noguchi, Kenji Chamoto, Zhang Yue, Kazunori Fugo, Harumichi Ishigame, Yoichiro Iwakura, Hidemitsu Kitamura, Takashi Nishimura IL-6-dependent spontaneous proliferation is required for the induction of colitogenic IL-17-producing CD8+ T cells. IL-6-dependent spontaneous proliferation is required for the induction of colitogenic IL-17-producing CD8+ T cells. IL-6-dependent spontaneous proliferation is required for the induction of colitogenic IL-17-producing CD8+ T cells. The Journal of experimental medicine, 205, 5, 1019-27 The Journal of experimental medicine, 205, 5, 1019-27 The Journal of experimental medicine, 205, 5, 1019-27 2008/05/12 English Research paper(scientific journal) Disclose to all
Daisuke Noguchi, Daiko Wakita, Masaki Tajima, Shigeru Ashino, Yoichiro Iwakura, Yue Zhang, Kenji Chamoto, Hidemitsu Kitamura, Takashi Nishimura Daisuke Noguchi, Daiko Wakita, Masaki Tajima, Shigeru Ashino, Yoichiro Iwakura, Yue Zhang, Kenji Chamoto, Hidemitsu Kitamura, Takashi Nishimura Daisuke Noguchi, Daiko Wakita, Masaki Tajima, Shigeru Ashino, Yoichiro Iwakura, Yue Zhang, Kenji Chamoto, Hidemitsu Kitamura, Takashi Nishimura Blocking of IL-6 signaling pathway prevents CD4+ T cell-mediated colitis in a T(h)17-independent manner. Blocking of IL-6 signaling pathway prevents CD4+ T cell-mediated colitis in a T(h)17-independent manner. Blocking of IL-6 signaling pathway prevents CD4+ T cell-mediated colitis in a T(h)17-independent manner. International immunology, 19, 12, 1431-40 International immunology, 19, 12, 1431-40 International immunology, 19, 12, 1431-40 2007/12 English Research paper(scientific journal) Disclose to all
Title language:
External funds: competitive funds and Grants-in-Aid for Scientific Research (Kakenhi)
Type Position Title(Japanese) Title(English) Period
若手研究 Representative PD-1阻害による抗腫瘍効果の最大化を目指したTr2細胞の応用性の解析 (2020年度分) 2020/04/01-2021/03/31