樽本 雄介

Last Update: 2021/07/27 17:16:57

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Name(Kanji/Kana/Abecedarium Latinum)
樽本 雄介/タルモト ユウスケ/Yusuke Tarumoto
Primary Affiliation(Org1/Job title)
Institute for Frontier Life and Medical Sciences/Assistant Professor
Affiliated programs (koza)
Org1 Job title
Graduate Schools Medicine Assistant Professor
Graduate Schools Medicine Assistant Professor
Faculty
Org1 Job title
理学部
Contact Address
Type Address(Japanese) Address(English)
Office 京都市左京区聖護院河原町53 53 Kawahara-cho, Syogoin, Sakyo-ku, Kyoto
Phone
Type Number
Office 075-751-4100
E-mail Address
E-mail address
ytarumoto @ infront.kyoto-u.acjp
Academic Degree
Field(Japanese) Field(English) University(Japanese) University(English) Method
修士(生命科学) 京都大学 Kyoto University
博士(生命科学) PhD Life Science 京都大学 Kyoto University
ORCID ID
https://orcid.org/0000-0001-6652-9618
researchmap URL
https://researchmap.jp/7000008804
Research Topics
(Japanese)
多能性幹細胞における未分化性維持のための転写ネットワークの解明
(English)
Investigation of Transcriptional Network in Pluripotent Stem Cells
Published Papers
Author Author(Japanese) Author(English) Title Title(Japanese) Title(English) Bibliography Bibliography(Japanese) Bibliography(English) Publication date Refereed paper Language Publishing type Disclose
Yan Zi Au, Muxin Gu, Etienne De Braekeleer, Malgorzata Gozdecka, Demetrios Aspris, Yusuke Tarumoto, Jonathan Cooper, Jason Yu, Swee Hoe Ong, Xi Chen, Konstantinos Tzelepis, Brian J P Huntly, George Vassiliou, Kosuke Yusa Yan Zi Au, Muxin Gu, Etienne De Braekeleer, Malgorzata Gozdecka, Demetrios Aspris, Yusuke Tarumoto, Jonathan Cooper, Jason Yu, Swee Hoe Ong, Xi Chen, Konstantinos Tzelepis, Brian J P Huntly, George Vassiliou, Kosuke Yusa Yan Zi Au, Muxin Gu, Etienne De Braekeleer, Malgorzata Gozdecka, Demetrios Aspris, Yusuke Tarumoto, Jonathan Cooper, Jason Yu, Swee Hoe Ong, Xi Chen, Konstantinos Tzelepis, Brian J P Huntly, George Vassiliou, Kosuke Yusa KAT7 is a genetic vulnerability of acute myeloid leukemias driven by MLL rearrangements. KAT7 is a genetic vulnerability of acute myeloid leukemias driven by MLL rearrangements. KAT7 is a genetic vulnerability of acute myeloid leukemias driven by MLL rearrangements. Leukemia, 35, 4, 1012-1022 Leukemia, 35, 4, 1012-1022 Leukemia, 35, 4, 1012-1022 2021/04 Refereed English Research paper(scientific journal) Disclose to all
Sumiko Takao, Lauren Forbes, Masahiro Uni, Shuyuan Cheng, Jose Mario Bello Pineda, Yusuke Tarumoto, Paolo Cifani, Gerard Minuesa, Celine Chen, Michael G Kharas, Robert K Bradley, Christopher R Vakoc, Richard P Koche, Alex Kentsis Sumiko Takao, Lauren Forbes, Masahiro Uni, Shuyuan Cheng, Jose Mario Bello Pineda, Yusuke Tarumoto, Paolo Cifani, Gerard Minuesa, Celine Chen, Michael G Kharas, Robert K Bradley, Christopher R Vakoc, Richard P Koche, Alex Kentsis Sumiko Takao, Lauren Forbes, Masahiro Uni, Shuyuan Cheng, Jose Mario Bello Pineda, Yusuke Tarumoto, Paolo Cifani, Gerard Minuesa, Celine Chen, Michael G Kharas, Robert K Bradley, Christopher R Vakoc, Richard P Koche, Alex Kentsis Convergent organization of aberrant MYB complex controls oncogenic gene expression in acute myeloid leukemia. Convergent organization of aberrant MYB complex controls oncogenic gene expression in acute myeloid leukemia. Convergent organization of aberrant MYB complex controls oncogenic gene expression in acute myeloid leukemia. eLife, 10 eLife, 10 eLife, 10 2021/02/02 Refereed English Research paper(scientific journal) Disclose to all
Yusuke Tarumoto, Shan Lin, Jinhua Wang, Joseph P Milazzo, Yali Xu, Bin Lu, Zhaolin Yang, Yiliang Wei, Sofya Polyanskaya, Mark Wunderlich, Nathanael S Gray, Kimberly Stegmaier, Christopher R Vakoc Yusuke Tarumoto, Shan Lin, Jinhua Wang, Joseph P Milazzo, Yali Xu, Bin Lu, Zhaolin Yang, Yiliang Wei, Sofya Polyanskaya, Mark Wunderlich, Nathanael S Gray, Kimberly Stegmaier, Christopher R Vakoc Yusuke Tarumoto, Shan Lin, Jinhua Wang, Joseph P Milazzo, Yali Xu, Bin Lu, Zhaolin Yang, Yiliang Wei, Sofya Polyanskaya, Mark Wunderlich, Nathanael S Gray, Kimberly Stegmaier, Christopher R Vakoc Salt-inducible kinase inhibition suppresses acute myeloid leukemia progression in vivo. Salt-inducible kinase inhibition suppresses acute myeloid leukemia progression in vivo. Salt-inducible kinase inhibition suppresses acute myeloid leukemia progression in vivo. Blood, 135, 1, 56-70 Blood, 135, 1, 56-70 Blood, 135, 1, 56-70 2020/01/02 Refereed English Research paper(scientific journal) Disclose to all
Hiroyuki Irie, Io Yamamoto, Yusuke Tarumoto, Sanki Tashiro, Kurt W Runge, Fuyuki Ishikawa Hiroyuki Irie, Io Yamamoto, Yusuke Tarumoto, Sanki Tashiro, Kurt W Runge, Fuyuki Ishikawa Hiroyuki Irie, Io Yamamoto, Yusuke Tarumoto, Sanki Tashiro, Kurt W Runge, Fuyuki Ishikawa Telomere-binding proteins Taz1 and Rap1 regulate DSB repair and suppress gross chromosomal rearrangements in fission yeast. Telomere-binding proteins Taz1 and Rap1 regulate DSB repair and suppress gross chromosomal rearrangements in fission yeast. Telomere-binding proteins Taz1 and Rap1 regulate DSB repair and suppress gross chromosomal rearrangements in fission yeast. PLoS genetics, 15, 8, e1008335 PLoS genetics, 15, 8, e1008335 PLoS genetics, 15, 8, e1008335 2019/08 Refereed English Research paper(scientific journal) Disclose to all
Miguel Foronda, Yusuke Tarumoto, Emma M Schatoff, Benjamin I Leach, Bianca J Diaz, Jill Zimmerman, Sukanya Goswami, Michael Shusterman, Christopher R Vakoc, Lukas E Dow Miguel Foronda, Yusuke Tarumoto, Emma M Schatoff, Benjamin I Leach, Bianca J Diaz, Jill Zimmerman, Sukanya Goswami, Michael Shusterman, Christopher R Vakoc, Lukas E Dow Miguel Foronda, Yusuke Tarumoto, Emma M Schatoff, Benjamin I Leach, Bianca J Diaz, Jill Zimmerman, Sukanya Goswami, Michael Shusterman, Christopher R Vakoc, Lukas E Dow Tankyrase inhibition sensitizes cells to CDK4 blockade. Tankyrase inhibition sensitizes cells to CDK4 blockade. Tankyrase inhibition sensitizes cells to CDK4 blockade. PloS one, 14, 12, e0226645 PloS one, 14, 12, e0226645 PloS one, 14, 12, e0226645 2019 Refereed English Research paper(scientific journal) Disclose to all
Bin Lu, Olaf Klingbeil, Yusuke Tarumoto, Tim D D Somerville, Yu-Han Huang, Yiliang Wei, Dorothy C Wai, Jason K K Low, Joseph P Milazzo, Xiaoli S Wu, Zhendong Cao, Xiaomei Yan, Osama E Demerdash, Gang Huang, Joel P Mackay, Justin B Kinney, Junwei Shi, Christopher R Vakoc Bin Lu, Olaf Klingbeil, Yusuke Tarumoto, Tim D D Somerville, Yu-Han Huang, Yiliang Wei, Dorothy C Wai, Jason K K Low, Joseph P Milazzo, Xiaoli S Wu, Zhendong Cao, Xiaomei Yan, Osama E Demerdash, Gang Huang, Joel P Mackay, Justin B Kinney, Junwei Shi, Christopher R Vakoc Bin Lu, Olaf Klingbeil, Yusuke Tarumoto, Tim D D Somerville, Yu-Han Huang, Yiliang Wei, Dorothy C Wai, Jason K K Low, Joseph P Milazzo, Xiaoli S Wu, Zhendong Cao, Xiaomei Yan, Osama E Demerdash, Gang Huang, Joel P Mackay, Justin B Kinney, Junwei Shi, Christopher R Vakoc A Transcription Factor Addiction in Leukemia Imposed by the MLL Promoter Sequence. A Transcription Factor Addiction in Leukemia Imposed by the MLL Promoter Sequence. A Transcription Factor Addiction in Leukemia Imposed by the MLL Promoter Sequence. Cancer cell, 34, 6, 970-981 Cancer cell, 34, 6, 970-981 Cancer cell, 34, 6, 970-981 2018/12/10 Refereed English Research paper(scientific journal) Disclose to all
Yusuke Tarumoto, Bin Lu, Tim D D Somerville, Yu-Han Huang, Joseph P Milazzo, Xiaoli S Wu, Olaf Klingbeil, Osama El Demerdash, Junwei Shi, Christopher R Vakoc Yusuke Tarumoto, Bin Lu, Tim D D Somerville, Yu-Han Huang, Joseph P Milazzo, Xiaoli S Wu, Olaf Klingbeil, Osama El Demerdash, Junwei Shi, Christopher R Vakoc Yusuke Tarumoto, Bin Lu, Tim D D Somerville, Yu-Han Huang, Joseph P Milazzo, Xiaoli S Wu, Olaf Klingbeil, Osama El Demerdash, Junwei Shi, Christopher R Vakoc LKB1, Salt-Inducible Kinases, and MEF2C Are Linked Dependencies in Acute Myeloid Leukemia. LKB1, Salt-Inducible Kinases, and MEF2C Are Linked Dependencies in Acute Myeloid Leukemia. LKB1, Salt-Inducible Kinases, and MEF2C Are Linked Dependencies in Acute Myeloid Leukemia. Molecular cell, 69, 6, 1017-1027 Molecular cell, 69, 6, 1017-1027 Molecular cell, 69, 6, 1017-1027 2018/03/15 Refereed English Research paper(scientific journal) Disclose to all
Yali Xu, Joseph P Milazzo, Tim D D Somerville, Yusuke Tarumoto, Yu-Han Huang, Elizabeth L Ostrander, John E Wilkinson, Grant A Challen, Christopher R Vakoc Yali Xu, Joseph P Milazzo, Tim D D Somerville, Yusuke Tarumoto, Yu-Han Huang, Elizabeth L Ostrander, John E Wilkinson, Grant A Challen, Christopher R Vakoc Yali Xu, Joseph P Milazzo, Tim D D Somerville, Yusuke Tarumoto, Yu-Han Huang, Elizabeth L Ostrander, John E Wilkinson, Grant A Challen, Christopher R Vakoc A TFIID-SAGA Perturbation that Targets MYB and Suppresses Acute Myeloid Leukemia. A TFIID-SAGA Perturbation that Targets MYB and Suppresses Acute Myeloid Leukemia. A TFIID-SAGA Perturbation that Targets MYB and Suppresses Acute Myeloid Leukemia. Cancer cell, 33, 1, 13-28 Cancer cell, 33, 1, 13-28 Cancer cell, 33, 1, 13-28 2018/01/08 Refereed English Research paper(scientific journal) Disclose to all
Masahiro Takikawa, Yusuke Tarumoto, Fuyuki Ishikawa Masahiro Takikawa, Yusuke Tarumoto, Fuyuki Ishikawa Masahiro Takikawa, Yusuke Tarumoto, Fuyuki Ishikawa Fission yeast Stn1 is crucial for semi-conservative replication at telomeres and subtelomeres. Fission yeast Stn1 is crucial for semi-conservative replication at telomeres and subtelomeres. Fission yeast Stn1 is crucial for semi-conservative replication at telomeres and subtelomeres. Nucleic acids research, 45, 3, 1255-1269 Nucleic acids research, 45, 3, 1255-1269 Nucleic acids research, 45, 3, 1255-1269 2017/02/17 Refereed English Research paper(scientific journal) Disclose to all
Yusuke Tarumoto, Junko Kanoh, Fuyuki Ishikawa Yusuke Tarumoto, Junko Kanoh, Fuyuki Ishikawa Yusuke Tarumoto, Junko Kanoh, Fuyuki Ishikawa Receptor for activated C-kinase (RACK1) homolog Cpc2 facilitates the general amino acid control response through Gcn2 kinase in fission yeast. Receptor for activated C-kinase (RACK1) homolog Cpc2 facilitates the general amino acid control response through Gcn2 kinase in fission yeast. Receptor for activated C-kinase (RACK1) homolog Cpc2 facilitates the general amino acid control response through Gcn2 kinase in fission yeast. The Journal of biological chemistry, 288, 26, 19260-8 The Journal of biological chemistry, 288, 26, 19260-8 The Journal of biological chemistry, 288, 26, 19260-8 2013/06/28 Refereed English Research paper(scientific journal) Disclose to all
Moeko Chujo, Yusuke Tarumoto, Koichi Miyatake, Eisuke Nishida, Fuyuki Ishikawa Moeko Chujo, Yusuke Tarumoto, Koichi Miyatake, Eisuke Nishida, Fuyuki Ishikawa Moeko Chujo, Yusuke Tarumoto, Koichi Miyatake, Eisuke Nishida, Fuyuki Ishikawa HIRA, a conserved histone chaperone, plays an essential role in low-dose stress response via transcriptional stimulation in fission yeast. HIRA, a conserved histone chaperone, plays an essential role in low-dose stress response via transcriptional stimulation in fission yeast. HIRA, a conserved histone chaperone, plays an essential role in low-dose stress response via transcriptional stimulation in fission yeast. The Journal of biological chemistry, 287, 28, 23440-50 The Journal of biological chemistry, 287, 28, 23440-50 The Journal of biological chemistry, 287, 28, 23440-50 2012/07/06 Refereed English Research paper(scientific journal) Disclose to all
Harutake Yamazaki, Yusuke Tarumoto, Fuyuki Ishikawa Harutake Yamazaki, Yusuke Tarumoto, Fuyuki Ishikawa Harutake Yamazaki, Yusuke Tarumoto, Fuyuki Ishikawa Tel1(ATM) and Rad3(ATR) phosphorylate the telomere protein Ccq1 to recruit telomerase and elongate telomeres in fission yeast. Tel1(ATM) and Rad3(ATR) phosphorylate the telomere protein Ccq1 to recruit telomerase and elongate telomeres in fission yeast. Tel1(ATM) and Rad3(ATR) phosphorylate the telomere protein Ccq1 to recruit telomerase and elongate telomeres in fission yeast. Genes & development, 26, 3, 241-6 Genes & development, 26, 3, 241-6 Genes & development, 26, 3, 241-6 2012/02/01 Refereed English Research paper(scientific journal) Disclose to all

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Title language:
Misc
Author Author(Japanese) Author(English) Title Title(Japanese) Title(English) Bibliography Bibliography(Japanese) Bibliography(English) Publication date Refereed paper Language Publishing type Disclose
樽本雄介, 遊佐宏介 樽本雄介, 遊佐宏介 樽本雄介, 遊佐宏介 Development and improvement of high-throughput CRISPR-Cas9 genetic screening CRISPR/Cas9による遺伝学的スクリーニングシステムの開発 Development and improvement of high-throughput CRISPR-Cas9 genetic screening 生体の科学, 71, 6 生体の科学, 71, 6 生体の科学, 71, 6 2020 Disclose to all
樽本雄介 樽本雄介 樽本雄介 Chemical inhibition of SIK3 as a therapeutic strategy in acute myeloid leukemia. SIK3抑制による急性骨髄性白血病の制御 Chemical inhibition of SIK3 as a therapeutic strategy in acute myeloid leukemia. 月刊血液内科, 81, 4, 529-534 月刊血液内科, 81, 4, 529-534 月刊血液内科, 81, 4, 529-534 2020 Japanese Disclose to all
長垣 良和, 中條 萌絵子, 樽本 雄介, 八尾 良司, 石川 冬木 長垣 良和, 中條 萌絵子, 樽本 雄介, 八尾 良司, 石川 冬木 長垣 良和, 中條 萌絵子, 樽本 雄介, 八尾 良司, 石川 冬木 哺乳類細胞の低容量ストレス応答におけるヒストンシャペロンHIRAの機能解析(Roles of histone chaperone HIRA in low-dose stress responses in mammalian cells) 哺乳類細胞の低容量ストレス応答におけるヒストンシャペロンHIRAの機能解析(Roles of histone chaperone HIRA in low-dose stress responses in mammalian cells) 哺乳類細胞の低容量ストレス応答におけるヒストンシャペロンHIRAの機能解析(Roles of histone chaperone HIRA in low-dose stress responses in mammalian cells) 日本癌学会総会記事, 73回, J-3079 日本癌学会総会記事, 73回, J-3079 日本癌学会総会記事, 73回, J-3079 2014/09 English Disclose to all
滝川雅大, 樽本雄介, 石川冬木 滝川雅大, 樽本雄介, 石川冬木 分裂酵母Stn1の染色体構造維持における役割 分裂酵母Stn1の染色体構造維持における役割 日本分子生物学会年会プログラム・要旨集(Web), 37th 日本分子生物学会年会プログラム・要旨集(Web), 37th , 37th 2014 Disclose to all
Yusuke Tarumoto, Fuyuki Ishikawa 樽本 雄介, 石川冬木 Yusuke Tarumoto, Fuyuki Ishikawa Cellular response to low-dose stress 低容量ストレスに対する細胞応答 Cellular response to low-dose stress Leading Author’s, 3, e004 領域融合レビュー, 3, e004 Leading Author’s, 3, e004 2014 Refereed Japanese Article, review, commentary, editorial, etc.(trade magazine, newspaper, online media) Disclose to all
入江 裕之, 樽本 雄介, 石川 冬木 入江 裕之, 樽本 雄介, 石川 冬木 入江 裕之, 樽本 雄介, 石川 冬木 分裂酵母テロメア結合タンパク質による大規模染色体再編の制御(Increased rates of gross chromosomal rearrangements in fission yeast cells defective for telomere-binding proteins) 分裂酵母テロメア結合タンパク質による大規模染色体再編の制御(Increased rates of gross chromosomal rearrangements in fission yeast cells defective for telomere-binding proteins) 分裂酵母テロメア結合タンパク質による大規模染色体再編の制御(Increased rates of gross chromosomal rearrangements in fission yeast cells defective for telomere-binding proteins) 日本癌学会総会記事, 72回, 120-120 日本癌学会総会記事, 72回, 120-120 日本癌学会総会記事, 72回, 120-120 2013/10 English Disclose to all
樽本雄介, 加納純子, 加納純子, 上野敏秀, 間野博行, 間野博行, 石川冬木 樽本雄介, 加納純子, 加納純子, 上野敏秀, 間野博行, 間野博行, 石川冬木 分裂酵母Cpc2はストレス応答性MAPキナーゼを介したストレス応答を促進する 分裂酵母Cpc2はストレス応答性MAPキナーゼを介したストレス応答を促進する 日本生化学会大会(Web), 86th 日本生化学会大会(Web), 86th , 86th 2013 Disclose to all
入江裕之, 樽本雄介, 石川冬木 入江裕之, 樽本雄介, 石川冬木 分裂酵母テロメア結合タンパク質による大規模染色体再編の制御 分裂酵母テロメア結合タンパク質による大規模染色体再編の制御 日本分子生物学会年会プログラム・要旨集(Web), 35th 日本分子生物学会年会プログラム・要旨集(Web), 35th , 35th 2012 Disclose to all
樽本雄介, 加納純子, 加納純子, 上野敏秀, 間野博行, 間野博行, 石川冬木 樽本雄介, 加納純子, 加納純子, 上野敏秀, 間野博行, 間野博行, 石川冬木 分裂酵母Cpc2はストレス応答性MAPキナーゼを介したストレス応答を促進する 分裂酵母Cpc2はストレス応答性MAPキナーゼを介したストレス応答を促進する 日本分子生物学会年会プログラム・要旨集(Web), 35th, 2P-251 日本分子生物学会年会プログラム・要旨集(Web), 35th, 2P-251 , 35th, 2P-251 2012 Japanese Disclose to all
二宮 源紀, 三河 拓己, 樽本 雄介, 石川 冬木 二宮 源紀, 三河 拓己, 樽本 雄介, 石川 冬木 二宮 源紀, 三河 拓己, 樽本 雄介, 石川 冬木 分裂酵母におけるチェックポイント活性化延長に対する細胞応答(Cellular responses to prolonged checkpoint activation in fission yeast) 分裂酵母におけるチェックポイント活性化延長に対する細胞応答(Cellular responses to prolonged checkpoint activation in fission yeast) 分裂酵母におけるチェックポイント活性化延長に対する細胞応答(Cellular responses to prolonged checkpoint activation in fission yeast) 日本生化学会大会・日本分子生物学会年会合同大会講演要旨集, 83回・33回, 3P-0365 日本生化学会大会・日本分子生物学会年会合同大会講演要旨集, 83回・33回, 3P-0365 日本生化学会大会・日本分子生物学会年会合同大会講演要旨集, 83回・33回, 3P-0365 2010/12 English Disclose to all
樽本雄介, 加納純子, 加納純子, 石川冬木 樽本雄介, 加納純子, 加納純子, 石川冬木 Cpc2/RACK1は環境からのストレスに対する交差耐性に機能する Cpc2/RACK1は環境からのストレスに対する交差耐性に機能する 生化学 生化学 2009 Disclose to all
樽本雄介, 加納純子, 石川冬木 樽本雄介, 加納純子, 石川冬木 Cpc2/RACK1は環境からのストレスに対する交差耐性に機能する Cpc2/RACK1は環境からのストレスに対する交差耐性に機能する 日本RNA学会年会要旨集, 11th, 3T6a-4 日本RNA学会年会要旨集, 11th, 3T6a-4 , 11th, 3T6a-4 2009 Japanese Disclose to all
樽本雄介, 加納純子, 石川冬木 樽本雄介, 加納純子, 石川冬木 分裂酵母Cpc2はeIF2αキナーゼによるストレス応答に関与する 分裂酵母Cpc2はeIF2αキナーゼによるストレス応答に関与する 生化学 生化学 2007 Disclose to all

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Title language:
Conference Activities & Talks
Title Title(Japanese) Title(English) Conference Conference(Japanese) Conference(English) Promotor Promotor(Japanese) Promotor(English) Date Language Assortment Disclose
がん細胞の転写ネットワーク依存性とその制御 がん細胞の転写ネットワーク依存性とその制御 京都大学ウイルス・再生医科学研究所 第4回個体の中の細胞社会学セミナー 京都大学ウイルス・再生医科学研究所 第4回個体の中の細胞社会学セミナー 2019/05/23 Japanese Public discourse, seminar, tutorial, course, lecture and others Disclose to all
機能ドメインを標的としたCRISPRスクリーニングによるがんの新たな治療標的の探索 機能ドメインを標的としたCRISPRスクリーニングによるがんの新たな治療標的の探索 東京大学医科学研究所 学友会セミナー 東京大学医科学研究所 学友会セミナー 2018/12/05 Japanese Public discourse, seminar, tutorial, course, lecture and others Disclose to all
機能ドメインを標的としたCRISPRスクリーニングによるがんの新たな治療標的の探索 機能ドメインを標的としたCRISPRスクリーニングによるがんの新たな治療標的の探索 金沢大学がん進展制御研究所セミナー 金沢大学がん進展制御研究所セミナー 2018/12/03 Japanese Public discourse, seminar, tutorial, course, lecture and others Disclose to all
Identification of cancer drug target by functional domain-focused CRISPR screening 機能的なタンパク質ドメインに対するCRISPRスクリーニングによるがんの治療標的の探索 Identification of cancer drug target by functional domain-focused CRISPR screening The 41st Annual Meeting of the Molecular Biology Society of Japan 第41回日本分子生物学会年会 The 41st Annual Meeting of the Molecular Biology Society of Japan 2018/11/28 Japanese Poster presentation Disclose to all
Targeting transcriptional vulnerability by linked dependency in acute myeloid leukemia Targeting transcriptional vulnerability by linked dependency in acute myeloid leukemia Targeting transcriptional vulnerability by linked dependency in acute myeloid leukemia 京都大学 生命科学セミナー 京都大学 生命科学セミナー 2018/09/14 English Public discourse, seminar, tutorial, course, lecture and others Disclose to all
Chemical inhibition of SIK3 as a therapeutic strategy in MEF2C-addicted acute myeloid leukemia Chemical inhibition of SIK3 as a therapeutic strategy in MEF2C-addicted acute myeloid leukemia Chemical inhibition of SIK3 as a therapeutic strategy in MEF2C-addicted acute myeloid leukemia CSHL Mechanisms & Models of Cancer CSHL Mechanisms & Models of Cancer CSHL Mechanisms & Models of Cancer 2018/08/15 English Oral presentation(general) Disclose to all
Cpc2 facilitates stress-activated MAP kinase-mediated stress response Cpc2 facilitates stress-activated MAP kinase-mediated stress response Cpc2 facilitates stress-activated MAP kinase-mediated stress response EMBO Conference on Fission Yeast: pombe 2013 EMBO Conference on Fission Yeast: pombe 2013 EMBO Conference on Fission Yeast: pombe 2013 2013/06/27 English Oral presentation(general) Disclose to all
Fission yeast Cpc2 facilitates stress-induced transcriptional and translational responses[Invited] Fission yeast Cpc2 facilitates stress-induced transcriptional and translational responses [Invited] Fission yeast Cpc2 facilitates stress-induced transcriptional and translational responses [Invited] The Sixth International Fission Yeast Meeting The Sixth International Fission Yeast Meeting The Sixth International Fission Yeast Meeting 2011/06/28 English Oral presentation(invited, special) Disclose to all
Title language:
External funds: competitive funds and Grants-in-Aid for Scientific Research (Kakenhi)
Type Position Title(Japanese) Title(English) Period
若手研究 Representative 転写抑制補因子による多能性幹細胞の未分化制御機構 (2020年度分) 2020/04/01-2021/03/31
若手研究 Representative 転写抑制補因子による多能性幹細胞の未分化制御機構 2020-2022
若手研究(B) Representative ストレス交差耐性におけるクロマチン構造変化と遺伝子発現制御 2013-2015
挑戦的萌芽研究 Assignment 分裂酵母を用いた条件的ヘテロクロマチン制御の解析 2011-2012
基盤研究(S) Assignment 発がんにおけるテロメア機能 2010-2015
External funds: other than those above
System Main person Title(Japanese) Title(English) Period
Lauri Strauss Leukemia Foundation Grant 樽本雄介 Targeting Linked Dependency Between Kinase and Transcription Factor 2018-2018
Lauri Strauss Leukemia Foundation Grant 樽本雄介 Identification of Genetic Dependencies and Therapeutic Targets in AML 2016-2017
Lauri Strauss Leukemia Foundation Grant 樽本雄介 Investigation of mechanism of epigenetic inhibitor-induced therapeutic activity 2015-2016
京都大学若手研究者スタートアップ研究費 樽本雄介 低容量ストレス応答の分子機構の解析 2009-2010
Teaching subject(s)
Name(Japanese) Name(English) Term Department Period
生物学実習A Laboratory work in Biological Science A 前期 理学部 2014/04-2015/03
生物科学課題研究21 Special Study CourseII(Biological Sciences) 21 通年 理学部 2014/04-2015/03
Faculty management (title, position)
Title Period
化学物質管理委員会 2019/10/01-