中島沙恵子

Last Update: 2018/06/15 11:58:30

Name(Kanji/Kana/Abecedarium Latinum)

中島沙恵子/ナカジマサエコ/Saeko Nakajima

Primary department(Org1/Job title)

Kyoto University Hospital/Assistant Professor

Contact address

Type	Address(Japanese)	Address(English)
Office	京都市左京区聖護院川原町５４	54 Shogoin Kawahara, Sakyo, Kyoto Japan 606-8507

Affiliated academic organization(s) (Japanese)

Organization name(Japanese)	Organization name(English)
日本皮膚科学会
日本研究皮膚科学会
日本免疫学会
日本炎症再生医学会
日本皮膚アレルギー・接触皮膚炎学会
日本アレルギー学会

Affiliated academic organization(s) (overseas)

Organization name	Country
Society of investigative dermatology	USA

Academic degree

Field(Japanese)	Field(English)	University(Japanese)	University(English)	Method
博士（医学）		京都大学

Graduate school

University(Japanese)	University(English)	Faculty(Japanese)	Faculty(English)	Major(Japanese)	Major(English)	Degree
京都大学		大学院医学研究科博士課程内科系専攻				修了

University

University(Japanese)	University(English)	Faculty(Japanese)	Faculty(English)	Major(s)(Japanese)	Major(s)(English)	Degree
大阪医科大学		医学部医学科				卒業

Research Topics

(Japanese)
炎症性皮膚疾患、皮膚常在微生物
(English)
Inflammatory skin diseases, skin microbiome

Published Papers

Author	Author(Japanese)	Author(English)	Title	Title(Japanese)	Title(English)	Bibliography	Bibliography(Japanese)	Bibliography(English)	Publication date	Refereed paper	Disclose
Ueharaguchi Y, Honda T, Kusuba N, Hanakawa S, Adachi A, Sawada Y, Otsuka A, Kitoh A, Dainichi T, Egawa G, Nakashima C, Nakajima S, Murata T, Ono S, Arita M, Narumiya S, Miyachi Y, Kabashima K	Ueharaguchi Y, Honda T, Kusuba N, Hanakawa S, Adachi A, Sawada Y, Otsuka A, Kitoh A, Dainichi T, Egawa G, Nakashima C, Nakajima S, Murata T, Ono S, Arita M, Narumiya S, Miyachi Y, Kabashima K	Ueharaguchi Y, Honda T, Kusuba N, Hanakawa S, Adachi A, Sawada Y, Otsuka A, Kitoh A, Dainichi T, Egawa G, Nakashima C, Nakajima S, Murata T, Ono S, Arita M, Narumiya S, Miyachi Y, Kabashima K	Thromboxane A<sub>2</sub> facilitates IL-17A production from Vγ4<sup>+</sup> γδ T cells and promotes psoriatic dermatitis in mice.	Thromboxane A<sub>2</sub> facilitates IL-17A production from Vγ4<sup>+</sup> γδ T cells and promotes psoriatic dermatitis in mice.	Thromboxane A<sub>2</sub> facilitates IL-17A production from Vγ4<sup>+</sup> γδ T cells and promotes psoriatic dermatitis in mice.	The Journal of allergy and clinical immunology	The Journal of allergy and clinical immunology	The Journal of allergy and clinical immunology	2018/04	Refereed	Disclose to all
Wen HC, Czarnowicki T, Noda S, Malik K, Pavel AB, Nakajima S, Honda T, Shin JU, Lee H, Chou M, Estrada Y, Zheng X, Xu H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E	Wen HC, Czarnowicki T, Noda S, Malik K, Pavel AB, Nakajima S, Honda T, Shin JU, Lee H, Chou M, Estrada Y, Zheng X, Xu H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E	Wen HC, Czarnowicki T, Noda S, Malik K, Pavel AB, Nakajima S, Honda T, Shin JU, Lee H, Chou M, Estrada Y, Zheng X, Xu H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E	Serum from Asian patients with atopic dermatitis is characterized by T<sub>H</sub>2/T<sub>H</sub>22 activation, which is highly correlated with nonlesional skin measures.	Serum from Asian patients with atopic dermatitis is characterized by T<sub>H</sub>2/T<sub>H</sub>22 activation, which is highly correlated with nonlesional skin measures.	Serum from Asian patients with atopic dermatitis is characterized by T<sub>H</sub>2/T<sub>H</sub>22 activation, which is highly correlated with nonlesional skin measures.	The Journal of allergy and clinical immunology	The Journal of allergy and clinical immunology	The Journal of allergy and clinical immunology	2018/04	Refereed	Disclose to all
Kusuba N, Kitoh A, Dainichi T, Honda T, Otsuka A, Egawa G, Nakajima S, Miyachi Y, Kabashima K	Kusuba N, Kitoh A, Dainichi T, Honda T, Otsuka A, Egawa G, Nakajima S, Miyachi Y, Kabashima K	Kusuba N, Kitoh A, Dainichi T, Honda T, Otsuka A, Egawa G, Nakajima S, Miyachi Y, Kabashima K	Inhibition of IL-17-committed T cells in a murine psoriasis model by a vitamin D analogue.	Inhibition of IL-17-committed T cells in a murine psoriasis model by a vitamin D analogue.	Inhibition of IL-17-committed T cells in a murine psoriasis model by a vitamin D analogue.	The Journal of allergy and clinical immunology, 141, 3, 972-981.e10	The Journal of allergy and clinical immunology, 141, 3, 972-981.e10	The Journal of allergy and clinical immunology, 141, 3, 972-981.e10	2018/03	Refereed	Disclose to all
Ono S, Egawa G, Kitoh A, Dainichi T, Otsuka A, Nakajima S, Honda T, Kabashima K	Ono S, Egawa G, Kitoh A, Dainichi T, Otsuka A, Nakajima S, Honda T, Kabashima K	Ono S, Egawa G, Kitoh A, Dainichi T, Otsuka A, Nakajima S, Honda T, Kabashima K	Local inflammation exacerbates cutaneous manifestations in a murine autoimmune pemphigus model.	Local inflammation exacerbates cutaneous manifestations in a murine autoimmune pemphigus model.	Local inflammation exacerbates cutaneous manifestations in a murine autoimmune pemphigus model.	The Journal of allergy and clinical immunology, 139, 6, 2026-2028.e5	The Journal of allergy and clinical immunology, 139, 6, 2026-2028.e5	The Journal of allergy and clinical immunology, 139, 6, 2026-2028.e5	2017/06	Refereed	Disclose to all
Honda T, Yamamoto O, Sawada Y, Egawa G, Kitoh A, Otsuka A, Dainichi T, Nakajima S, Miyachi Y, Kabashima K	Honda T, Yamamoto O, Sawada Y, Egawa G, Kitoh A, Otsuka A, Dainichi T, Nakajima S, Miyachi Y, Kabashima K	Honda T, Yamamoto O, Sawada Y, Egawa G, Kitoh A, Otsuka A, Dainichi T, Nakajima S, Miyachi Y, Kabashima K	Receptor-interacting protein kinase 3 controls keratinocyte activation in a necroptosis-independent manner and promotes psoriatic dermatitis in mice.	Receptor-interacting protein kinase 3 controls keratinocyte activation in a necroptosis-independent manner and promotes psoriatic dermatitis in mice.	Receptor-interacting protein kinase 3 controls keratinocyte activation in a necroptosis-independent manner and promotes psoriatic dermatitis in mice.	The Journal of allergy and clinical immunology, 140, 2, 619-622.e6	The Journal of allergy and clinical immunology, 140, 2, 619-622.e6	The Journal of allergy and clinical immunology, 140, 2, 619-622.e6	2017/03	Refereed	Disclose to all
Ewald DA, Noda S, Oliva M, Litman T, Nakajima S, Li X, Xu H, Workman CT, Scheipers P, Svitacheva N, Labuda T, Krueger JG, Suárez-Fariñas M, Kabashima K, Guttman-Yassky E	Ewald DA, Noda S, Oliva M, Litman T, Nakajima S, Li X, Xu H, Workman CT, Scheipers P, Svitacheva N, Labuda T, Krueger JG, Suárez-Fariñas M, Kabashima K, Guttman-Yassky E	Ewald DA, Noda S, Oliva M, Litman T, Nakajima S, Li X, Xu H, Workman CT, Scheipers P, Svitacheva N, Labuda T, Krueger JG, Suárez-Fariñas M, Kabashima K, Guttman-Yassky E	Major differences between human atopic dermatitis and murine models, as determined by using global transcriptomic profiling.	Major differences between human atopic dermatitis and murine models, as determined by using global transcriptomic profiling.	Major differences between human atopic dermatitis and murine models, as determined by using global transcriptomic profiling.	The Journal of allergy and clinical immunology, 139, 2, 562-571	The Journal of allergy and clinical immunology, 139, 2, 562-571	The Journal of allergy and clinical immunology, 139, 2, 562-571	2017/02	Refereed	Disclose to all
Rerknimitr P, Tanizaki H, Yamamoto Y, Amano W, Nakajima S, Nakashima C, Nonomura Y, Wititsuwannakul J, Miyachi Y, Otsuka A, Kabashima K	Rerknimitr P, Tanizaki H, Yamamoto Y, Amano W, Nakajima S, Nakashima C, Nonomura Y, Wititsuwannakul J, Miyachi Y, Otsuka A, Kabashima K	Rerknimitr P, Tanizaki H, Yamamoto Y, Amano W, Nakajima S, Nakashima C, Nonomura Y, Wititsuwannakul J, Miyachi Y, Otsuka A, Kabashima K	Decreased Filaggrin Level May Lead to Sweat Duct Obstruction in Filaggrin Mutant Mice.	Decreased Filaggrin Level May Lead to Sweat Duct Obstruction in Filaggrin Mutant Mice.	Decreased Filaggrin Level May Lead to Sweat Duct Obstruction in Filaggrin Mutant Mice.	The Journal of investigative dermatology, 137, 1, 248-251	The Journal of investigative dermatology, 137, 1, 248-251	The Journal of investigative dermatology, 137, 1, 248-251	2017/01	Refereed	Disclose to all
Kuroda E, Ozasa K, Temizoz B, Ohata K, Koo CX, Kanuma T, Kusakabe T, Kobari S, Horie M, Morimoto Y, Nakajima S, Kabashima K, Ziegler SF, Iwakura Y, Ise W, Kurosaki T, Nagatake T, Kunisawa J, Takemura N, Uematsu S, Hayashi M, Aoshi T, Kobiyama K, Coban C, Ishii KJ	Kuroda E, Ozasa K, Temizoz B, Ohata K, Koo CX, Kanuma T, Kusakabe T, Kobari S, Horie M, Morimoto Y, Nakajima S, Kabashima K, Ziegler SF, Iwakura Y, Ise W, Kurosaki T, Nagatake T, Kunisawa J, Takemura N, Uematsu S, Hayashi M, Aoshi T, Kobiyama K, Coban C, Ishii KJ	Kuroda E, Ozasa K, Temizoz B, Ohata K, Koo CX, Kanuma T, Kusakabe T, Kobari S, Horie M, Morimoto Y, Nakajima S, Kabashima K, Ziegler SF, Iwakura Y, Ise W, Kurosaki T, Nagatake T, Kunisawa J, Takemura N, Uematsu S, Hayashi M, Aoshi T, Kobiyama K, Coban C, Ishii KJ	Inhaled Fine Particles Induce Alveolar Macrophage Death and Interleukin-1α Release to Promote Inducible Bronchus-Associated Lymphoid Tissue Formation.	Inhaled Fine Particles Induce Alveolar Macrophage Death and Interleukin-1α Release to Promote Inducible Bronchus-Associated Lymphoid Tissue Formation.	Inhaled Fine Particles Induce Alveolar Macrophage Death and Interleukin-1α Release to Promote Inducible Bronchus-Associated Lymphoid Tissue Formation.	Immunity, 45, 6, 1299-1310	Immunity, 45, 6, 1299-1310	Immunity, 45, 6, 1299-1310	2016/12	Refereed	Disclose to all
Amano W, Nakajima S, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K	Amano W, Nakajima S, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K	Amano W, Nakajima S, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K	JAK inhibitor JTE-052 regulates contact hypersensitivity by downmodulating T cell activation and differentiation.	JAK inhibitor JTE-052 regulates contact hypersensitivity by downmodulating T cell activation and differentiation.	JAK inhibitor JTE-052 regulates contact hypersensitivity by downmodulating T cell activation and differentiation.	Journal of dermatological science, 84, 3, 265-265	Journal of dermatological science, 84, 3, 265-265	Journal of dermatological science, 84, 3, 265-265	2016/12	Refereed	Disclose to all
Wong LS, Otsuka A, Yamamoto Y, Nonomura Y, Nakashima C, Honda T, Dainichi T, Kitoh A, Nakajima S, Hirakawa S, Miyachi Y, Kabashima K	Wong LS, Otsuka A, Yamamoto Y, Nonomura Y, Nakashima C, Honda T, Dainichi T, Kitoh A, Nakajima S, Hirakawa S, Miyachi Y, Kabashima K	Wong LS, Otsuka A, Yamamoto Y, Nonomura Y, Nakashima C, Honda T, Dainichi T, Kitoh A, Nakajima S, Hirakawa S, Miyachi Y, Kabashima K	Vascular endothelial growth factor partially induces pruritus via epidermal hyperinnervation in imiquimod-induced psoriasiform dermatitis in mice.	Vascular endothelial growth factor partially induces pruritus via epidermal hyperinnervation in imiquimod-induced psoriasiform dermatitis in mice.	Vascular endothelial growth factor partially induces pruritus via epidermal hyperinnervation in imiquimod-induced psoriasiform dermatitis in mice.	Journal of dermatological science, 83, 2, 148-151	Journal of dermatological science, 83, 2, 148-151	Journal of dermatological science, 83, 2, 148-151	2016/08	Refereed	Disclose to all
Sasaki H, Otsuka A, Iga N, Rerknimitr P, Nakajima S, Kaku Y, Sakurai T, Dainichi T, Kabashima K	Sasaki H, Otsuka A, Iga N, Rerknimitr P, Nakajima S, Kaku Y, Sakurai T, Dainichi T, Kabashima K	Sasaki H, Otsuka A, Iga N, Rerknimitr P, Nakajima S, Kaku Y, Sakurai T, Dainichi T, Kabashima K	A case of rosacea fulminans staining negative for LL-37.	A case of rosacea fulminans staining negative for LL-37.	A case of rosacea fulminans staining negative for LL-37.	European journal of dermatology : EJD, 26, 1, 101-102	European journal of dermatology : EJD, 26, 1, 101-102	European journal of dermatology : EJD, 26, 1, 101-102	2016/01	Refereed	Disclose to all
Nonomura Y, Otsuka A, Nakashima C, Seidel JA, Kitoh A, Dainichi T, Nakajima S, Sawada Y, Matsushita S, Aoki M, Takenouchi T, Fujimura T, Hatta N, Koreeda S, Fukushima S, Honda T, Kabashima K	Nonomura Y, Otsuka A, Nakashima C, Seidel JA, Kitoh A, Dainichi T, Nakajima S, Sawada Y, Matsushita S, Aoki M, Takenouchi T, Fujimura T, Hatta N, Koreeda S, Fukushima S, Honda T, Kabashima K	Nonomura Y, Otsuka A, Nakashima C, Seidel JA, Kitoh A, Dainichi T, Nakajima S, Sawada Y, Matsushita S, Aoki M, Takenouchi T, Fujimura T, Hatta N, Koreeda S, Fukushima S, Honda T, Kabashima K	Peripheral blood Th9 cells are a possible pharmacodynamic biomarker of nivolumab treatment efficacy in metastatic melanoma patients.	Peripheral blood Th9 cells are a possible pharmacodynamic biomarker of nivolumab treatment efficacy in metastatic melanoma patients.	Peripheral blood Th9 cells are a possible pharmacodynamic biomarker of nivolumab treatment efficacy in metastatic melanoma patients.	Oncoimmunology, 5, 12, e1248327	Oncoimmunology, 5, 12, e1248327	Oncoimmunology, 5, 12, e1248327	2016	Refereed	Disclose to all
Noda S, Suárez-Fariñas M, Ungar B, Kim SJ, de Guzman Strong C, Xu H, Peng X, Estrada YD, Nakajima S, Honda T, Shin JU, Lee H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E	Noda S, Suárez-Fariñas M, Ungar B, Kim SJ, de Guzman Strong C, Xu H, Peng X, Estrada YD, Nakajima S, Honda T, Shin JU, Lee H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E	Noda S, Suárez-Fariñas M, Ungar B, Kim SJ, de Guzman Strong C, Xu H, Peng X, Estrada YD, Nakajima S, Honda T, Shin JU, Lee H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E	The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization.	The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization.	The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization.	The Journal of allergy and clinical immunology, 136, 5, 1254-1264	The Journal of allergy and clinical immunology, 136, 5, 1254-1264	The Journal of allergy and clinical immunology, 136, 5, 1254-1264	2015/11	Refereed	Disclose to all
Sawada Y, Honda T, Hanakawa S, Nakamizo S, Murata T, Ueharaguchi-Tanada Y, Ono S, Amano W, Nakajima S, Egawa G, Tanizaki H, Otsuka A, Kitoh A, Dainichi T, Ogawa N, Kobayashi Y, Yokomizo T, Arita M, Nakamura M, Miyachi Y, Kabashima K	Sawada Y, Honda T, Hanakawa S, Nakamizo S, Murata T, Ueharaguchi-Tanada Y, Ono S, Amano W, Nakajima S, Egawa G, Tanizaki H, Otsuka A, Kitoh A, Dainichi T, Ogawa N, Kobayashi Y, Yokomizo T, Arita M, Nakamura M, Miyachi Y, Kabashima K	Sawada Y, Honda T, Hanakawa S, Nakamizo S, Murata T, Ueharaguchi-Tanada Y, Ono S, Amano W, Nakajima S, Egawa G, Tanizaki H, Otsuka A, Kitoh A, Dainichi T, Ogawa N, Kobayashi Y, Yokomizo T, Arita M, Nakamura M, Miyachi Y, Kabashima K	Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses.	Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses.	Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses.	The Journal of experimental medicine, 212, 11, 1921-1930	The Journal of experimental medicine, 212, 11, 1921-1930	The Journal of experimental medicine, 212, 11, 1921-1930	2015/10	Refereed	Disclose to all
Amano W, Nakajima S, Kunugi H, Numata Y, Kitoh A, Egawa G, Dainichi T, Honda T, Otsuka A, Kimoto Y, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K	Amano W, Nakajima S, Kunugi H, Numata Y, Kitoh A, Egawa G, Dainichi T, Honda T, Otsuka A, Kimoto Y, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K	Amano W, Nakajima S, Kunugi H, Numata Y, Kitoh A, Egawa G, Dainichi T, Honda T, Otsuka A, Kimoto Y, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K	The Janus kinase inhibitor JTE-052 improves skin barrier function through suppressing signal transducer and activator of transcription 3 signaling.	The Janus kinase inhibitor JTE-052 improves skin barrier function through suppressing signal transducer and activator of transcription 3 signaling.	The Janus kinase inhibitor JTE-052 improves skin barrier function through suppressing signal transducer and activator of transcription 3 signaling.	The Journal of allergy and clinical immunology, 136, 3, 667-677.e7	The Journal of allergy and clinical immunology, 136, 3, 667-677.e7	The Journal of allergy and clinical immunology, 136, 3, 667-677.e7	2015/09	Refereed	Disclose to all
Nakamizo S, Egawa G, Tomura M, Sakai S, Tsuchiya S, Kitoh A, Honda T, Otsuka A, Nakajima S, Dainichi T, Tanizaki H, Mitsuyama M, Sugimoto Y, Kawai K, Yoshikai Y, Miyachi Y, Kabashima K	Nakamizo S, Egawa G, Tomura M, Sakai S, Tsuchiya S, Kitoh A, Honda T, Otsuka A, Nakajima S, Dainichi T, Tanizaki H, Mitsuyama M, Sugimoto Y, Kawai K, Yoshikai Y, Miyachi Y, Kabashima K	Nakamizo S, Egawa G, Tomura M, Sakai S, Tsuchiya S, Kitoh A, Honda T, Otsuka A, Nakajima S, Dainichi T, Tanizaki H, Mitsuyama M, Sugimoto Y, Kawai K, Yoshikai Y, Miyachi Y, Kabashima K	Dermal Vγ4(+) γδ T cells possess a migratory potency to the draining lymph nodes and modulate CD8(+) T-cell activity through TNF-α production.	Dermal Vγ4(+) γδ T cells possess a migratory potency to the draining lymph nodes and modulate CD8(+) T-cell activity through TNF-α production.	Dermal Vγ4(+) γδ T cells possess a migratory potency to the draining lymph nodes and modulate CD8(+) T-cell activity through TNF-α production.	The Journal of investigative dermatology, 135, 4, 1007-1015	The Journal of investigative dermatology, 135, 4, 1007-1015	The Journal of investigative dermatology, 135, 4, 1007-1015	2015/04	Refereed	Disclose to all
Shibuya R, Tanizaki H, Nakajima S, Koyanagi I, Kataoka TR, Miyachi Y, Kabashima K	Shibuya R, Tanizaki H, Nakajima S, Koyanagi I, Kataoka TR, Miyachi Y, Kabashima K	Shibuya R, Tanizaki H, Nakajima S, Koyanagi I, Kataoka TR, Miyachi Y, Kabashima K	DIHS/DRESS with remarkable eosinophilic pneumonia caused by zonisamide.	DIHS/DRESS with remarkable eosinophilic pneumonia caused by zonisamide.	DIHS/DRESS with remarkable eosinophilic pneumonia caused by zonisamide.	Acta dermato-venereologica, 95, 2, 229-230	Acta dermato-venereologica, 95, 2, 229-230	Acta dermato-venereologica, 95, 2, 229-230	2015/02	Refereed	Disclose to all
Nakamizo S, Egawa G, Honda T, Nakajima S, Belkaid Y, Kabashima K	Nakamizo S, Egawa G, Honda T, Nakajima S, Belkaid Y, Kabashima K	Nakamizo S, Egawa G, Honda T, Nakajima S, Belkaid Y, Kabashima K	Commensal bacteria and cutaneous immunity.	Commensal bacteria and cutaneous immunity.	Commensal bacteria and cutaneous immunity.	Seminars in immunopathology, 37, 1, 73-80	Seminars in immunopathology, 37, 1, 73-80	Seminars in immunopathology, 37, 1, 73-80	2015/01	Refereed	Disclose to all
Natsuaki Y, Egawa G, Nakamizo S, Ono S, Hanakawa S, Okada T, Kusuba N, Otsuka A, Kitoh A, Honda T, Nakajima S, Tsuchiya S, Sugimoto Y, Ishii KJ, Tsutsui H, Yagita H, Iwakura Y, Kubo M, Ng Lg, Hashimoto T, Fuentes J, Guttman-Yassky E, Miyachi Y, Kabashima K	Natsuaki Y, Egawa G, Nakamizo S, Ono S, Hanakawa S, Okada T, Kusuba N, Otsuka A, Kitoh A, Honda T, Nakajima S, Tsuchiya S, Sugimoto Y, Ishii KJ, Tsutsui H, Yagita H, Iwakura Y, Kubo M, Ng Lg, Hashimoto T, Fuentes J, Guttman-Yassky E, Miyachi Y, Kabashima K	Natsuaki Y, Egawa G, Nakamizo S, Ono S, Hanakawa S, Okada T, Kusuba N, Otsuka A, Kitoh A, Honda T, Nakajima S, Tsuchiya S, Sugimoto Y, Ishii KJ, Tsutsui H, Yagita H, Iwakura Y, Kubo M, Ng Lg, Hashimoto T, Fuentes J, Guttman-Yassky E, Miyachi Y, Kabashima K	Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin.	Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin.	Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin.	Nature immunology, 15, 11, 1064-1069	Nature immunology, 15, 11, 1064-1069	Nature immunology, 15, 11, 1064-1069	2014/11	Refereed	Disclose to all
Shimizuhira C, Otsuka A, Honda T, Kitoh A, Egawa G, Nakajima S, Nakashima C, Watarai H, Miyachi Y, Kabashima K	Shimizuhira C, Otsuka A, Honda T, Kitoh A, Egawa G, Nakajima S, Nakashima C, Watarai H, Miyachi Y, Kabashima K	Shimizuhira C, Otsuka A, Honda T, Kitoh A, Egawa G, Nakajima S, Nakashima C, Watarai H, Miyachi Y, Kabashima K	Natural killer T cells are essential for the development of contact hypersensitivity in BALB/c mice.	Natural killer T cells are essential for the development of contact hypersensitivity in BALB/c mice.	Natural killer T cells are essential for the development of contact hypersensitivity in BALB/c mice.	The Journal of investigative dermatology, 134, 11, 2709-2718	The Journal of investigative dermatology, 134, 11, 2709-2718	The Journal of investigative dermatology, 134, 11, 2709-2718	2014/11	Refereed	Disclose to all
Nakajima S, Kitoh A, Egawa G, Natsuaki Y, Nakamizo S, Moniaga CS, Otsuka A, Honda T, Hanakawa S, Amano W, Iwakura Y, Nakae S, Kubo M, Miyachi Y, Kabashima K	Nakajima S, Kitoh A, Egawa G, Natsuaki Y, Nakamizo S, Moniaga CS, Otsuka A, Honda T, Hanakawa S, Amano W, Iwakura Y, Nakae S, Kubo M, Miyachi Y, Kabashima K	Nakajima S, Kitoh A, Egawa G, Natsuaki Y, Nakamizo S, Moniaga CS, Otsuka A, Honda T, Hanakawa S, Amano W, Iwakura Y, Nakae S, Kubo M, Miyachi Y, Kabashima K	IL-17A as an inducer for Th2 immune responses in murine atopic dermatitis models.	IL-17A as an inducer for Th2 immune responses in murine atopic dermatitis models.	IL-17A as an inducer for Th2 immune responses in murine atopic dermatitis models.	The Journal of investigative dermatology, 134, 8, 2122-2130	The Journal of investigative dermatology, 134, 8, 2122-2130	The Journal of investigative dermatology, 134, 8, 2122-2130	2014/08	Refereed	Disclose to all
Nakashima C, Otsuka A, Kitoh A, Honda T, Egawa G, Nakajima S, Nakamizo S, Arita M, Kubo M, Miyachi Y, Kabashima K	Nakashima C, Otsuka A, Kitoh A, Honda T, Egawa G, Nakajima S, Nakamizo S, Arita M, Kubo M, Miyachi Y, Kabashima K	Nakashima C, Otsuka A, Kitoh A, Honda T, Egawa G, Nakajima S, Nakamizo S, Arita M, Kubo M, Miyachi Y, Kabashima K	Basophils regulate the recruitment of eosinophils in a murine model of irritant contact dermatitis.	Basophils regulate the recruitment of eosinophils in a murine model of irritant contact dermatitis.	Basophils regulate the recruitment of eosinophils in a murine model of irritant contact dermatitis.	The Journal of allergy and clinical immunology, 134, 1, 100-107	The Journal of allergy and clinical immunology, 134, 1, 100-107	The Journal of allergy and clinical immunology, 134, 1, 100-107	2014/07	Refereed	Disclose to all
Otsuka A, Doi H, Egawa G, Maekawa A, Fujita T, Nakamizo S, Nakashima C, Nakajima S, Watanabe T, Miyachi Y, Narumiya S, Kabashima K	Otsuka A, Doi H, Egawa G, Maekawa A, Fujita T, Nakamizo S, Nakashima C, Nakajima S, Watanabe T, Miyachi Y, Narumiya S, Kabashima K	Otsuka A, Doi H, Egawa G, Maekawa A, Fujita T, Nakamizo S, Nakashima C, Nakajima S, Watanabe T, Miyachi Y, Narumiya S, Kabashima K	Possible new therapeutic strategy to regulate atopic dermatitis through upregulating filaggrin expression.	Possible new therapeutic strategy to regulate atopic dermatitis through upregulating filaggrin expression.	Possible new therapeutic strategy to regulate atopic dermatitis through upregulating filaggrin expression.	The Journal of allergy and clinical immunology, 133, 1, 139-146	The Journal of allergy and clinical immunology, 133, 1, 139-146	The Journal of allergy and clinical immunology, 133, 1, 139-146	2014/01	Refereed	Disclose to all
Ono S, Nakajima S, Otsuka A, Miyachi Y, Kabashima K	Ono S, Nakajima S, Otsuka A, Miyachi Y, Kabashima K	Ono S, Nakajima S, Otsuka A, Miyachi Y, Kabashima K	Pigmented purpuric dermatitis with high expression levels of serum TARC/CCL17 and epidermal TSLP.	Pigmented purpuric dermatitis with high expression levels of serum TARC/CCL17 and epidermal TSLP.	Pigmented purpuric dermatitis with high expression levels of serum TARC/CCL17 and epidermal TSLP.	European journal of dermatology : EJD, 23, 5, 701-702	European journal of dermatology : EJD, 23, 5, 701-702	European journal of dermatology : EJD, 23, 5, 701-702	2013/09	Refereed	Disclose to all
Mori T, Kabashima K, Fukamachi S, Kuroda E, Sakabe J, Kobayashi M, Nakajima S, Nakano K, Tanaka Y, Matsushita S, Nakamura M, Tokura Y	Mori T, Kabashima K, Fukamachi S, Kuroda E, Sakabe J, Kobayashi M, Nakajima S, Nakano K, Tanaka Y, Matsushita S, Nakamura M, Tokura Y	Mori T, Kabashima K, Fukamachi S, Kuroda E, Sakabe J, Kobayashi M, Nakajima S, Nakano K, Tanaka Y, Matsushita S, Nakamura M, Tokura Y	D1-like dopamine receptors antagonist inhibits cutaneous immune reactions mediated by Th2 and mast cells.	D1-like dopamine receptors antagonist inhibits cutaneous immune reactions mediated by Th2 and mast cells.	D1-like dopamine receptors antagonist inhibits cutaneous immune reactions mediated by Th2 and mast cells.	Journal of dermatological science, 71, 1, 37-44	Journal of dermatological science, 71, 1, 37-44	Journal of dermatological science, 71, 1, 37-44	2013/07	Refereed	Disclose to all
Moniaga CS, Jeong SK, Egawa G, Nakajima S, Hara-Chikuma M, Jeon JE, Lee SH, Hibino T, Miyachi Y, Kabashima K	Moniaga CS, Jeong SK, Egawa G, Nakajima S, Hara-Chikuma M, Jeon JE, Lee SH, Hibino T, Miyachi Y, Kabashima K	Moniaga CS, Jeong SK, Egawa G, Nakajima S, Hara-Chikuma M, Jeon JE, Lee SH, Hibino T, Miyachi Y, Kabashima K	Protease activity enhances production of thymic stromal lymphopoietin and basophil accumulation in flaky tail mice.	Protease activity enhances production of thymic stromal lymphopoietin and basophil accumulation in flaky tail mice.	Protease activity enhances production of thymic stromal lymphopoietin and basophil accumulation in flaky tail mice.	The American journal of pathology, 182, 3, 841-851	The American journal of pathology, 182, 3, 841-851	The American journal of pathology, 182, 3, 841-851	2013/03	Refereed	Disclose to all
Shiraishi N, Nomura T, Tanizaki H, Nakajima S, Narumiya S, Miyachi Y, Tokura Y, Kabashima K	Shiraishi N, Nomura T, Tanizaki H, Nakajima S, Narumiya S, Miyachi Y, Tokura Y, Kabashima K	Shiraishi N, Nomura T, Tanizaki H, Nakajima S, Narumiya S, Miyachi Y, Tokura Y, Kabashima K	Prostaglandin E2-EP3 axis in fine-tuning excessive skin inflammation by restricting dendritic cell functions.	Prostaglandin E2-EP3 axis in fine-tuning excessive skin inflammation by restricting dendritic cell functions.	Prostaglandin E2-EP3 axis in fine-tuning excessive skin inflammation by restricting dendritic cell functions.	PloS one, 8, 7	PloS one, 8, 7	PloS one, 8, 7	2013	Refereed	Disclose to all
Otsuka A, Nakajima S, Kubo M, Egawa G, Honda T, Kitoh A, Nomura T, Hanakawa S, Sagita Moniaga C, Kim B, Matsuoka S, Watanabe T, Miyachi Y, Kabashima K	Otsuka A, Nakajima S, Kubo M, Egawa G, Honda T, Kitoh A, Nomura T, Hanakawa S, Sagita Moniaga C, Kim B, Matsuoka S, Watanabe T, Miyachi Y, Kabashima K	Otsuka A, Nakajima S, Kubo M, Egawa G, Honda T, Kitoh A, Nomura T, Hanakawa S, Sagita Moniaga C, Kim B, Matsuoka S, Watanabe T, Miyachi Y, Kabashima K	Basophils are required for the induction of Th2 immunity to haptens and peptide antigens.	Basophils are required for the induction of Th2 immunity to haptens and peptide antigens.	Basophils are required for the induction of Th2 immunity to haptens and peptide antigens.	Nature communications, 4	Nature communications, 4	Nature communications, 4	2013	Refereed	Disclose to all
Nakajima S, Igyártó BZ, Honda T, Egawa G, Otsuka A, Hara-Chikuma M, Watanabe N, Ziegler SF, Tomura M, Inaba K, Miyachi Y, Kaplan DH, Kabashima K	Nakajima S, Igyártó BZ, Honda T, Egawa G, Otsuka A, Hara-Chikuma M, Watanabe N, Ziegler SF, Tomura M, Inaba K, Miyachi Y, Kaplan DH, Kabashima K	Nakajima S, Igyártó BZ, Honda T, Egawa G, Otsuka A, Hara-Chikuma M, Watanabe N, Ziegler SF, Tomura M, Inaba K, Miyachi Y, Kaplan DH, Kabashima K	Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling.	Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling.	Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling.	The Journal of allergy and clinical immunology, 129, 4, 1048-1055	The Journal of allergy and clinical immunology, 129, 4, 1048-1055	The Journal of allergy and clinical immunology, 129, 4, 1048-1055	2012/04	Refereed	Disclose to all
Nakajima S, Watanabe H, Tohyama M, Sugita K, Iijima M, Hashimoto K, Tokura Y, Nishimura Y, Doi H, Tanioka M, Miyachi Y, Kabashima K	Nakajima S, Watanabe H, Tohyama M, Sugita K, Iijima M, Hashimoto K, Tokura Y, Nishimura Y, Doi H, Tanioka M, Miyachi Y, Kabashima K	Nakajima S, Watanabe H, Tohyama M, Sugita K, Iijima M, Hashimoto K, Tokura Y, Nishimura Y, Doi H, Tanioka M, Miyachi Y, Kabashima K	High-mobility group box 1 protein (HMGB1) as a novel diagnostic tool for toxic epidermal necrolysis and Stevens-Johnson syndrome.	High-mobility group box 1 protein (HMGB1) as a novel diagnostic tool for toxic epidermal necrolysis and Stevens-Johnson syndrome.	High-mobility group box 1 protein (HMGB1) as a novel diagnostic tool for toxic epidermal necrolysis and Stevens-Johnson syndrome.	Archives of dermatology, 147, 9, 1110-1112	Archives of dermatology, 147, 9, 1110-1112	Archives of dermatology, 147, 9, 1110-1112	2011/09	Refereed	Disclose to all
Otsuka A, Kubo M, Honda T, Egawa G, Nakajima S, Tanizaki H, Kim B, Matsuoka S, Watanabe T, Nakae S, Miyachi Y, Kabashima K	Otsuka A, Kubo M, Honda T, Egawa G, Nakajima S, Tanizaki H, Kim B, Matsuoka S, Watanabe T, Nakae S, Miyachi Y, Kabashima K	Otsuka A, Kubo M, Honda T, Egawa G, Nakajima S, Tanizaki H, Kim B, Matsuoka S, Watanabe T, Nakae S, Miyachi Y, Kabashima K	Requirement of interaction between mast cells and skin dendritic cells to establish contact hypersensitivity.	Requirement of interaction between mast cells and skin dendritic cells to establish contact hypersensitivity.	Requirement of interaction between mast cells and skin dendritic cells to establish contact hypersensitivity.	PloS one, 6, 9	PloS one, 6, 9	PloS one, 6, 9	2011	Refereed	Disclose to all
Tanizaki H, Egawa G, Inaba K, Honda T, Nakajima S, Moniaga CS, Otsuka A, Ishizaki T, Tomura M, Watanabe T, Miyachi Y, Narumiya S, Okada T, Kabashima K	Tanizaki H, Egawa G, Inaba K, Honda T, Nakajima S, Moniaga CS, Otsuka A, Ishizaki T, Tomura M, Watanabe T, Miyachi Y, Narumiya S, Okada T, Kabashima K	Tanizaki H, Egawa G, Inaba K, Honda T, Nakajima S, Moniaga CS, Otsuka A, Ishizaki T, Tomura M, Watanabe T, Miyachi Y, Narumiya S, Okada T, Kabashima K	Rho-mDia1 pathway is required for adhesion, migration, and T-cell stimulation in dendritic cells.	Rho-mDia1 pathway is required for adhesion, migration, and T-cell stimulation in dendritic cells.	Rho-mDia1 pathway is required for adhesion, migration, and T-cell stimulation in dendritic cells.	Blood, 116, 26, 5875-5884	Blood, 116, 26, 5875-5884	Blood, 116, 26, 5875-5884	2010/12	Refereed	Disclose to all
Sugita K, Tohyama M, Watanabe H, Otsuka A, Nakajima S, Iijima M, Hashimoto K, Tokura Y, Miyachi Y, Kabashima K	Sugita K, Tohyama M, Watanabe H, Otsuka A, Nakajima S, Iijima M, Hashimoto K, Tokura Y, Miyachi Y, Kabashima K	Sugita K, Tohyama M, Watanabe H, Otsuka A, Nakajima S, Iijima M, Hashimoto K, Tokura Y, Miyachi Y, Kabashima K	Fluctuation of blood and skin plasmacytoid dendritic cells in drug-induced hypersensitivity syndrome.	Fluctuation of blood and skin plasmacytoid dendritic cells in drug-induced hypersensitivity syndrome.	Fluctuation of blood and skin plasmacytoid dendritic cells in drug-induced hypersensitivity syndrome.	The Journal of allergy and clinical immunology, 126, 2, 408-410	The Journal of allergy and clinical immunology, 126, 2, 408-410	The Journal of allergy and clinical immunology, 126, 2, 408-410	2010/08	Refereed	Disclose to all
Nakajima S, Ohshima K, Kyogoku M, Miyachi Y, Kabashima K	Nakajima S, Ohshima K, Kyogoku M, Miyachi Y, Kabashima K	Nakajima S, Ohshima K, Kyogoku M, Miyachi Y, Kabashima K	A case of intravascular large B-cell lymphoma with atypical clinical manifestations and analysis of CXCL12 and CXCR4 expression.	A case of intravascular large B-cell lymphoma with atypical clinical manifestations and analysis of CXCL12 and CXCR4 expression.	A case of intravascular large B-cell lymphoma with atypical clinical manifestations and analysis of CXCL12 and CXCR4 expression.	Archives of dermatology, 146, 6, 686-687	Archives of dermatology, 146, 6, 686-687	Archives of dermatology, 146, 6, 686-687	2010/06	Refereed	Disclose to all
Nakajima S, Honda T, Sakata D, Egawa G, Tanizaki H, Otsuka A, Moniaga CS, Watanabe T, Miyachi Y, Narumiya S, Kabashima K	Nakajima S, Honda T, Sakata D, Egawa G, Tanizaki H, Otsuka A, Moniaga CS, Watanabe T, Miyachi Y, Narumiya S, Kabashima K	Nakajima S, Honda T, Sakata D, Egawa G, Tanizaki H, Otsuka A, Moniaga CS, Watanabe T, Miyachi Y, Narumiya S, Kabashima K	Prostaglandin I2-IP signaling promotes Th1 differentiation in a mouse model of contact hypersensitivity.	Prostaglandin I2-IP signaling promotes Th1 differentiation in a mouse model of contact hypersensitivity.	Prostaglandin I2-IP signaling promotes Th1 differentiation in a mouse model of contact hypersensitivity.	Journal of immunology (Baltimore, Md. : 1950), 184, 10, 5595-5603	Journal of immunology (Baltimore, Md. : 1950), 184, 10, 5595-5603	Journal of immunology (Baltimore, Md. : 1950), 184, 10, 5595-5603	2010/05	Refereed	Disclose to all
Moniaga CS, Egawa G, Kawasaki H, Hara-Chikuma M, Honda T, Tanizaki H, Nakajima S, Otsuka A, Matsuoka H, Kubo A, Sakabe J, Tokura Y, Miyachi Y, Amagai M, Kabashima K	Moniaga CS, Egawa G, Kawasaki H, Hara-Chikuma M, Honda T, Tanizaki H, Nakajima S, Otsuka A, Matsuoka H, Kubo A, Sakabe J, Tokura Y, Miyachi Y, Amagai M, Kabashima K	Moniaga CS, Egawa G, Kawasaki H, Hara-Chikuma M, Honda T, Tanizaki H, Nakajima S, Otsuka A, Matsuoka H, Kubo A, Sakabe J, Tokura Y, Miyachi Y, Amagai M, Kabashima K	Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract.	Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract.	Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract.	The American journal of pathology, 176, 5, 2385-2393	The American journal of pathology, 176, 5, 2385-2393	The American journal of pathology, 176, 5, 2385-2393	2010/05	Refereed	Disclose to all
Honda T, Nakajima S, Egawa G, Ogasawara K, Malissen B, Miyachi Y, Kabashima K	Honda T, Nakajima S, Egawa G, Ogasawara K, Malissen B, Miyachi Y, Kabashima K	Honda T, Nakajima S, Egawa G, Ogasawara K, Malissen B, Miyachi Y, Kabashima K	Compensatory role of Langerhans cells and langerin-positive dermal dendritic cells in the sensitization phase of murine contact hypersensitivity.	Compensatory role of Langerhans cells and langerin-positive dermal dendritic cells in the sensitization phase of murine contact hypersensitivity.	Compensatory role of Langerhans cells and langerin-positive dermal dendritic cells in the sensitization phase of murine contact hypersensitivity.	The Journal of allergy and clinical immunology, 125, 5, 1154-1156	The Journal of allergy and clinical immunology, 125, 5, 1154-1156	The Journal of allergy and clinical immunology, 125, 5, 1154-1156	2010/05	Refereed	Disclose to all
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History of staying abroad

Organization	Department	Research theme	Country	Period
National Institute of Allergic and Infectious Diseases	Laboratory of Parasitic Diseases/Mucosal Immunity Section	Skin commensals and host immunity	United States of America	2015/04/01-2017/03/31

中島 沙恵子

中島沙恵子