中島 沙恵子

Last Update: 2018/06/15 11:58:30

Print

Name(Kanji/Kana/Abecedarium Latinum)
中島 沙恵子/ナカジマ サエコ/Saeko Nakajima
Primary department(Org1/Job title)
Kyoto University Hospital/Assistant Professor
Contact address
Type Address(Japanese) Address(English)
Office 京都市左京区聖護院川原町54 54 Shogoin Kawahara, Sakyo, Kyoto Japan 606-8507
Affiliated academic organization(s) (Japanese)
Organization name(Japanese) Organization name(English)
日本皮膚科学会
日本研究皮膚科学会
日本免疫学会
日本炎症再生医学会
日本皮膚アレルギー・接触皮膚炎学会
日本アレルギー学会
Affiliated academic organization(s) (overseas)
Organization name Country
Society of investigative dermatology USA
Academic degree
Field(Japanese) Field(English) University(Japanese) University(English) Method
博士(医学) 京都大学
Graduate school
University(Japanese) University(English) Faculty(Japanese) Faculty(English) Major(Japanese) Major(English) Degree
京都大学 大学院医学研究科博士課程内科系専攻 修了
University
University(Japanese) University(English) Faculty(Japanese) Faculty(English) Major(s)(Japanese) Major(s)(English) Degree
大阪医科大学 医学部医学科 卒業
Research Topics
(Japanese)
炎症性皮膚疾患、皮膚常在微生物
(English)
Inflammatory skin diseases, skin microbiome
Published Papers
Author Author(Japanese) Author(English) Title Title(Japanese) Title(English) Bibliography Bibliography(Japanese) Bibliography(English) Publication date Refereed paper Language Publishing type Disclose
Ueharaguchi Y, Honda T, Kusuba N, Hanakawa S, Adachi A, Sawada Y, Otsuka A, Kitoh A, Dainichi T, Egawa G, Nakashima C, Nakajima S, Murata T, Ono S, Arita M, Narumiya S, Miyachi Y, Kabashima K Ueharaguchi Y, Honda T, Kusuba N, Hanakawa S, Adachi A, Sawada Y, Otsuka A, Kitoh A, Dainichi T, Egawa G, Nakashima C, Nakajima S, Murata T, Ono S, Arita M, Narumiya S, Miyachi Y, Kabashima K Ueharaguchi Y, Honda T, Kusuba N, Hanakawa S, Adachi A, Sawada Y, Otsuka A, Kitoh A, Dainichi T, Egawa G, Nakashima C, Nakajima S, Murata T, Ono S, Arita M, Narumiya S, Miyachi Y, Kabashima K Thromboxane A<sub>2</sub> facilitates IL-17A production from Vγ4<sup>+</sup> γδ T cells and promotes psoriatic dermatitis in mice. Thromboxane A<sub>2</sub> facilitates IL-17A production from Vγ4<sup>+</sup> γδ T cells and promotes psoriatic dermatitis in mice. Thromboxane A<sub>2</sub> facilitates IL-17A production from Vγ4<sup>+</sup> γδ T cells and promotes psoriatic dermatitis in mice. The Journal of allergy and clinical immunology The Journal of allergy and clinical immunology The Journal of allergy and clinical immunology 2018/04 Refereed Disclose to all
Wen HC, Czarnowicki T, Noda S, Malik K, Pavel AB, Nakajima S, Honda T, Shin JU, Lee H, Chou M, Estrada Y, Zheng X, Xu H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E Wen HC, Czarnowicki T, Noda S, Malik K, Pavel AB, Nakajima S, Honda T, Shin JU, Lee H, Chou M, Estrada Y, Zheng X, Xu H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E Wen HC, Czarnowicki T, Noda S, Malik K, Pavel AB, Nakajima S, Honda T, Shin JU, Lee H, Chou M, Estrada Y, Zheng X, Xu H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E Serum from Asian patients with atopic dermatitis is characterized by T<sub>H</sub>2/T<sub>H</sub>22 activation, which is highly correlated with nonlesional skin measures. Serum from Asian patients with atopic dermatitis is characterized by T<sub>H</sub>2/T<sub>H</sub>22 activation, which is highly correlated with nonlesional skin measures. Serum from Asian patients with atopic dermatitis is characterized by T<sub>H</sub>2/T<sub>H</sub>22 activation, which is highly correlated with nonlesional skin measures. The Journal of allergy and clinical immunology The Journal of allergy and clinical immunology The Journal of allergy and clinical immunology 2018/04 Refereed Disclose to all
Kusuba N, Kitoh A, Dainichi T, Honda T, Otsuka A, Egawa G, Nakajima S, Miyachi Y, Kabashima K Kusuba N, Kitoh A, Dainichi T, Honda T, Otsuka A, Egawa G, Nakajima S, Miyachi Y, Kabashima K Kusuba N, Kitoh A, Dainichi T, Honda T, Otsuka A, Egawa G, Nakajima S, Miyachi Y, Kabashima K Inhibition of IL-17-committed T cells in a murine psoriasis model by a vitamin D analogue. Inhibition of IL-17-committed T cells in a murine psoriasis model by a vitamin D analogue. Inhibition of IL-17-committed T cells in a murine psoriasis model by a vitamin D analogue. The Journal of allergy and clinical immunology, 141, 3, 972-981.e10 The Journal of allergy and clinical immunology, 141, 3, 972-981.e10 The Journal of allergy and clinical immunology, 141, 3, 972-981.e10 2018/03 Refereed Disclose to all
Ono S, Egawa G, Kitoh A, Dainichi T, Otsuka A, Nakajima S, Honda T, Kabashima K Ono S, Egawa G, Kitoh A, Dainichi T, Otsuka A, Nakajima S, Honda T, Kabashima K Ono S, Egawa G, Kitoh A, Dainichi T, Otsuka A, Nakajima S, Honda T, Kabashima K Local inflammation exacerbates cutaneous manifestations in a murine autoimmune pemphigus model. Local inflammation exacerbates cutaneous manifestations in a murine autoimmune pemphigus model. Local inflammation exacerbates cutaneous manifestations in a murine autoimmune pemphigus model. The Journal of allergy and clinical immunology, 139, 6, 2026-2028.e5 The Journal of allergy and clinical immunology, 139, 6, 2026-2028.e5 The Journal of allergy and clinical immunology, 139, 6, 2026-2028.e5 2017/06 Refereed Disclose to all
Honda T, Yamamoto O, Sawada Y, Egawa G, Kitoh A, Otsuka A, Dainichi T, Nakajima S, Miyachi Y, Kabashima K Honda T, Yamamoto O, Sawada Y, Egawa G, Kitoh A, Otsuka A, Dainichi T, Nakajima S, Miyachi Y, Kabashima K Honda T, Yamamoto O, Sawada Y, Egawa G, Kitoh A, Otsuka A, Dainichi T, Nakajima S, Miyachi Y, Kabashima K Receptor-interacting protein kinase 3 controls keratinocyte activation in a necroptosis-independent manner and promotes psoriatic dermatitis in mice. Receptor-interacting protein kinase 3 controls keratinocyte activation in a necroptosis-independent manner and promotes psoriatic dermatitis in mice. Receptor-interacting protein kinase 3 controls keratinocyte activation in a necroptosis-independent manner and promotes psoriatic dermatitis in mice. The Journal of allergy and clinical immunology, 140, 2, 619-622.e6 The Journal of allergy and clinical immunology, 140, 2, 619-622.e6 The Journal of allergy and clinical immunology, 140, 2, 619-622.e6 2017/03 Refereed Disclose to all
Ewald DA, Noda S, Oliva M, Litman T, Nakajima S, Li X, Xu H, Workman CT, Scheipers P, Svitacheva N, Labuda T, Krueger JG, Suárez-Fariñas M, Kabashima K, Guttman-Yassky E Ewald DA, Noda S, Oliva M, Litman T, Nakajima S, Li X, Xu H, Workman CT, Scheipers P, Svitacheva N, Labuda T, Krueger JG, Suárez-Fariñas M, Kabashima K, Guttman-Yassky E Ewald DA, Noda S, Oliva M, Litman T, Nakajima S, Li X, Xu H, Workman CT, Scheipers P, Svitacheva N, Labuda T, Krueger JG, Suárez-Fariñas M, Kabashima K, Guttman-Yassky E Major differences between human atopic dermatitis and murine models, as determined by using global transcriptomic profiling. Major differences between human atopic dermatitis and murine models, as determined by using global transcriptomic profiling. Major differences between human atopic dermatitis and murine models, as determined by using global transcriptomic profiling. The Journal of allergy and clinical immunology, 139, 2, 562-571 The Journal of allergy and clinical immunology, 139, 2, 562-571 The Journal of allergy and clinical immunology, 139, 2, 562-571 2017/02 Refereed Disclose to all
Rerknimitr P, Tanizaki H, Yamamoto Y, Amano W, Nakajima S, Nakashima C, Nonomura Y, Wititsuwannakul J, Miyachi Y, Otsuka A, Kabashima K Rerknimitr P, Tanizaki H, Yamamoto Y, Amano W, Nakajima S, Nakashima C, Nonomura Y, Wititsuwannakul J, Miyachi Y, Otsuka A, Kabashima K Rerknimitr P, Tanizaki H, Yamamoto Y, Amano W, Nakajima S, Nakashima C, Nonomura Y, Wititsuwannakul J, Miyachi Y, Otsuka A, Kabashima K Decreased Filaggrin Level May Lead to Sweat Duct Obstruction in Filaggrin Mutant Mice. Decreased Filaggrin Level May Lead to Sweat Duct Obstruction in Filaggrin Mutant Mice. Decreased Filaggrin Level May Lead to Sweat Duct Obstruction in Filaggrin Mutant Mice. The Journal of investigative dermatology, 137, 1, 248-251 The Journal of investigative dermatology, 137, 1, 248-251 The Journal of investigative dermatology, 137, 1, 248-251 2017/01 Refereed Disclose to all
Kuroda E, Ozasa K, Temizoz B, Ohata K, Koo CX, Kanuma T, Kusakabe T, Kobari S, Horie M, Morimoto Y, Nakajima S, Kabashima K, Ziegler SF, Iwakura Y, Ise W, Kurosaki T, Nagatake T, Kunisawa J, Takemura N, Uematsu S, Hayashi M, Aoshi T, Kobiyama K, Coban C, Ishii KJ Kuroda E, Ozasa K, Temizoz B, Ohata K, Koo CX, Kanuma T, Kusakabe T, Kobari S, Horie M, Morimoto Y, Nakajima S, Kabashima K, Ziegler SF, Iwakura Y, Ise W, Kurosaki T, Nagatake T, Kunisawa J, Takemura N, Uematsu S, Hayashi M, Aoshi T, Kobiyama K, Coban C, Ishii KJ Kuroda E, Ozasa K, Temizoz B, Ohata K, Koo CX, Kanuma T, Kusakabe T, Kobari S, Horie M, Morimoto Y, Nakajima S, Kabashima K, Ziegler SF, Iwakura Y, Ise W, Kurosaki T, Nagatake T, Kunisawa J, Takemura N, Uematsu S, Hayashi M, Aoshi T, Kobiyama K, Coban C, Ishii KJ Inhaled Fine Particles Induce Alveolar Macrophage Death and Interleukin-1α Release to Promote Inducible Bronchus-Associated Lymphoid Tissue Formation. Inhaled Fine Particles Induce Alveolar Macrophage Death and Interleukin-1α Release to Promote Inducible Bronchus-Associated Lymphoid Tissue Formation. Inhaled Fine Particles Induce Alveolar Macrophage Death and Interleukin-1α Release to Promote Inducible Bronchus-Associated Lymphoid Tissue Formation. Immunity, 45, 6, 1299-1310 Immunity, 45, 6, 1299-1310 Immunity, 45, 6, 1299-1310 2016/12 Refereed Disclose to all
Amano W, Nakajima S, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K Amano W, Nakajima S, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K Amano W, Nakajima S, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K JAK inhibitor JTE-052 regulates contact hypersensitivity by downmodulating T cell activation and differentiation. JAK inhibitor JTE-052 regulates contact hypersensitivity by downmodulating T cell activation and differentiation. JAK inhibitor JTE-052 regulates contact hypersensitivity by downmodulating T cell activation and differentiation. Journal of dermatological science, 84, 3, 265-265 Journal of dermatological science, 84, 3, 265-265 Journal of dermatological science, 84, 3, 265-265 2016/12 Refereed Disclose to all
Wong LS, Otsuka A, Yamamoto Y, Nonomura Y, Nakashima C, Honda T, Dainichi T, Kitoh A, Nakajima S, Hirakawa S, Miyachi Y, Kabashima K Wong LS, Otsuka A, Yamamoto Y, Nonomura Y, Nakashima C, Honda T, Dainichi T, Kitoh A, Nakajima S, Hirakawa S, Miyachi Y, Kabashima K Wong LS, Otsuka A, Yamamoto Y, Nonomura Y, Nakashima C, Honda T, Dainichi T, Kitoh A, Nakajima S, Hirakawa S, Miyachi Y, Kabashima K Vascular endothelial growth factor partially induces pruritus via epidermal hyperinnervation in imiquimod-induced psoriasiform dermatitis in mice. Vascular endothelial growth factor partially induces pruritus via epidermal hyperinnervation in imiquimod-induced psoriasiform dermatitis in mice. Vascular endothelial growth factor partially induces pruritus via epidermal hyperinnervation in imiquimod-induced psoriasiform dermatitis in mice. Journal of dermatological science, 83, 2, 148-151 Journal of dermatological science, 83, 2, 148-151 Journal of dermatological science, 83, 2, 148-151 2016/08 Refereed Disclose to all
Sasaki H, Otsuka A, Iga N, Rerknimitr P, Nakajima S, Kaku Y, Sakurai T, Dainichi T, Kabashima K Sasaki H, Otsuka A, Iga N, Rerknimitr P, Nakajima S, Kaku Y, Sakurai T, Dainichi T, Kabashima K Sasaki H, Otsuka A, Iga N, Rerknimitr P, Nakajima S, Kaku Y, Sakurai T, Dainichi T, Kabashima K A case of rosacea fulminans staining negative for LL-37. A case of rosacea fulminans staining negative for LL-37. A case of rosacea fulminans staining negative for LL-37. European journal of dermatology : EJD, 26, 1, 101-102 European journal of dermatology : EJD, 26, 1, 101-102 European journal of dermatology : EJD, 26, 1, 101-102 2016/01 Refereed Disclose to all
Nonomura Y, Otsuka A, Nakashima C, Seidel JA, Kitoh A, Dainichi T, Nakajima S, Sawada Y, Matsushita S, Aoki M, Takenouchi T, Fujimura T, Hatta N, Koreeda S, Fukushima S, Honda T, Kabashima K Nonomura Y, Otsuka A, Nakashima C, Seidel JA, Kitoh A, Dainichi T, Nakajima S, Sawada Y, Matsushita S, Aoki M, Takenouchi T, Fujimura T, Hatta N, Koreeda S, Fukushima S, Honda T, Kabashima K Nonomura Y, Otsuka A, Nakashima C, Seidel JA, Kitoh A, Dainichi T, Nakajima S, Sawada Y, Matsushita S, Aoki M, Takenouchi T, Fujimura T, Hatta N, Koreeda S, Fukushima S, Honda T, Kabashima K Peripheral blood Th9 cells are a possible pharmacodynamic biomarker of nivolumab treatment efficacy in metastatic melanoma patients. Peripheral blood Th9 cells are a possible pharmacodynamic biomarker of nivolumab treatment efficacy in metastatic melanoma patients. Peripheral blood Th9 cells are a possible pharmacodynamic biomarker of nivolumab treatment efficacy in metastatic melanoma patients. Oncoimmunology, 5, 12, e1248327 Oncoimmunology, 5, 12, e1248327 Oncoimmunology, 5, 12, e1248327 2016 Refereed Disclose to all
Noda S, Suárez-Fariñas M, Ungar B, Kim SJ, de Guzman Strong C, Xu H, Peng X, Estrada YD, Nakajima S, Honda T, Shin JU, Lee H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E Noda S, Suárez-Fariñas M, Ungar B, Kim SJ, de Guzman Strong C, Xu H, Peng X, Estrada YD, Nakajima S, Honda T, Shin JU, Lee H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E Noda S, Suárez-Fariñas M, Ungar B, Kim SJ, de Guzman Strong C, Xu H, Peng X, Estrada YD, Nakajima S, Honda T, Shin JU, Lee H, Krueger JG, Lee KH, Kabashima K, Guttman-Yassky E The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization. The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization. The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization. The Journal of allergy and clinical immunology, 136, 5, 1254-1264 The Journal of allergy and clinical immunology, 136, 5, 1254-1264 The Journal of allergy and clinical immunology, 136, 5, 1254-1264 2015/11 Refereed Disclose to all
Sawada Y, Honda T, Hanakawa S, Nakamizo S, Murata T, Ueharaguchi-Tanada Y, Ono S, Amano W, Nakajima S, Egawa G, Tanizaki H, Otsuka A, Kitoh A, Dainichi T, Ogawa N, Kobayashi Y, Yokomizo T, Arita M, Nakamura M, Miyachi Y, Kabashima K Sawada Y, Honda T, Hanakawa S, Nakamizo S, Murata T, Ueharaguchi-Tanada Y, Ono S, Amano W, Nakajima S, Egawa G, Tanizaki H, Otsuka A, Kitoh A, Dainichi T, Ogawa N, Kobayashi Y, Yokomizo T, Arita M, Nakamura M, Miyachi Y, Kabashima K Sawada Y, Honda T, Hanakawa S, Nakamizo S, Murata T, Ueharaguchi-Tanada Y, Ono S, Amano W, Nakajima S, Egawa G, Tanizaki H, Otsuka A, Kitoh A, Dainichi T, Ogawa N, Kobayashi Y, Yokomizo T, Arita M, Nakamura M, Miyachi Y, Kabashima K Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses. Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses. Resolvin E1 inhibits dendritic cell migration in the skin and attenuates contact hypersensitivity responses. The Journal of experimental medicine, 212, 11, 1921-1930 The Journal of experimental medicine, 212, 11, 1921-1930 The Journal of experimental medicine, 212, 11, 1921-1930 2015/10 Refereed Disclose to all
Amano W, Nakajima S, Kunugi H, Numata Y, Kitoh A, Egawa G, Dainichi T, Honda T, Otsuka A, Kimoto Y, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K Amano W, Nakajima S, Kunugi H, Numata Y, Kitoh A, Egawa G, Dainichi T, Honda T, Otsuka A, Kimoto Y, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K Amano W, Nakajima S, Kunugi H, Numata Y, Kitoh A, Egawa G, Dainichi T, Honda T, Otsuka A, Kimoto Y, Yamamoto Y, Tanimoto A, Matsushita M, Miyachi Y, Kabashima K The Janus kinase inhibitor JTE-052 improves skin barrier function through suppressing signal transducer and activator of transcription 3 signaling. The Janus kinase inhibitor JTE-052 improves skin barrier function through suppressing signal transducer and activator of transcription 3 signaling. The Janus kinase inhibitor JTE-052 improves skin barrier function through suppressing signal transducer and activator of transcription 3 signaling. The Journal of allergy and clinical immunology, 136, 3, 667-677.e7 The Journal of allergy and clinical immunology, 136, 3, 667-677.e7 The Journal of allergy and clinical immunology, 136, 3, 667-677.e7 2015/09 Refereed Disclose to all
Nakamizo S, Egawa G, Tomura M, Sakai S, Tsuchiya S, Kitoh A, Honda T, Otsuka A, Nakajima S, Dainichi T, Tanizaki H, Mitsuyama M, Sugimoto Y, Kawai K, Yoshikai Y, Miyachi Y, Kabashima K Nakamizo S, Egawa G, Tomura M, Sakai S, Tsuchiya S, Kitoh A, Honda T, Otsuka A, Nakajima S, Dainichi T, Tanizaki H, Mitsuyama M, Sugimoto Y, Kawai K, Yoshikai Y, Miyachi Y, Kabashima K Nakamizo S, Egawa G, Tomura M, Sakai S, Tsuchiya S, Kitoh A, Honda T, Otsuka A, Nakajima S, Dainichi T, Tanizaki H, Mitsuyama M, Sugimoto Y, Kawai K, Yoshikai Y, Miyachi Y, Kabashima K Dermal Vγ4(+) γδ T cells possess a migratory potency to the draining lymph nodes and modulate CD8(+) T-cell activity through TNF-α production. Dermal Vγ4(+) γδ T cells possess a migratory potency to the draining lymph nodes and modulate CD8(+) T-cell activity through TNF-α production. Dermal Vγ4(+) γδ T cells possess a migratory potency to the draining lymph nodes and modulate CD8(+) T-cell activity through TNF-α production. The Journal of investigative dermatology, 135, 4, 1007-1015 The Journal of investigative dermatology, 135, 4, 1007-1015 The Journal of investigative dermatology, 135, 4, 1007-1015 2015/04 Refereed Disclose to all
Shibuya R, Tanizaki H, Nakajima S, Koyanagi I, Kataoka TR, Miyachi Y, Kabashima K Shibuya R, Tanizaki H, Nakajima S, Koyanagi I, Kataoka TR, Miyachi Y, Kabashima K Shibuya R, Tanizaki H, Nakajima S, Koyanagi I, Kataoka TR, Miyachi Y, Kabashima K DIHS/DRESS with remarkable eosinophilic pneumonia caused by zonisamide. DIHS/DRESS with remarkable eosinophilic pneumonia caused by zonisamide. DIHS/DRESS with remarkable eosinophilic pneumonia caused by zonisamide. Acta dermato-venereologica, 95, 2, 229-230 Acta dermato-venereologica, 95, 2, 229-230 Acta dermato-venereologica, 95, 2, 229-230 2015/02 Refereed Disclose to all
Nakamizo S, Egawa G, Honda T, Nakajima S, Belkaid Y, Kabashima K Nakamizo S, Egawa G, Honda T, Nakajima S, Belkaid Y, Kabashima K Nakamizo S, Egawa G, Honda T, Nakajima S, Belkaid Y, Kabashima K Commensal bacteria and cutaneous immunity. Commensal bacteria and cutaneous immunity. Commensal bacteria and cutaneous immunity. Seminars in immunopathology, 37, 1, 73-80 Seminars in immunopathology, 37, 1, 73-80 Seminars in immunopathology, 37, 1, 73-80 2015/01 Refereed Disclose to all
Natsuaki Y, Egawa G, Nakamizo S, Ono S, Hanakawa S, Okada T, Kusuba N, Otsuka A, Kitoh A, Honda T, Nakajima S, Tsuchiya S, Sugimoto Y, Ishii KJ, Tsutsui H, Yagita H, Iwakura Y, Kubo M, Ng Lg, Hashimoto T, Fuentes J, Guttman-Yassky E, Miyachi Y, Kabashima K Natsuaki Y, Egawa G, Nakamizo S, Ono S, Hanakawa S, Okada T, Kusuba N, Otsuka A, Kitoh A, Honda T, Nakajima S, Tsuchiya S, Sugimoto Y, Ishii KJ, Tsutsui H, Yagita H, Iwakura Y, Kubo M, Ng Lg, Hashimoto T, Fuentes J, Guttman-Yassky E, Miyachi Y, Kabashima K Natsuaki Y, Egawa G, Nakamizo S, Ono S, Hanakawa S, Okada T, Kusuba N, Otsuka A, Kitoh A, Honda T, Nakajima S, Tsuchiya S, Sugimoto Y, Ishii KJ, Tsutsui H, Yagita H, Iwakura Y, Kubo M, Ng Lg, Hashimoto T, Fuentes J, Guttman-Yassky E, Miyachi Y, Kabashima K Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin. Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin. Perivascular leukocyte clusters are essential for efficient activation of effector T cells in the skin. Nature immunology, 15, 11, 1064-1069 Nature immunology, 15, 11, 1064-1069 Nature immunology, 15, 11, 1064-1069 2014/11 Refereed Disclose to all
Shimizuhira C, Otsuka A, Honda T, Kitoh A, Egawa G, Nakajima S, Nakashima C, Watarai H, Miyachi Y, Kabashima K Shimizuhira C, Otsuka A, Honda T, Kitoh A, Egawa G, Nakajima S, Nakashima C, Watarai H, Miyachi Y, Kabashima K Shimizuhira C, Otsuka A, Honda T, Kitoh A, Egawa G, Nakajima S, Nakashima C, Watarai H, Miyachi Y, Kabashima K Natural killer T cells are essential for the development of contact hypersensitivity in BALB/c mice. Natural killer T cells are essential for the development of contact hypersensitivity in BALB/c mice. Natural killer T cells are essential for the development of contact hypersensitivity in BALB/c mice. The Journal of investigative dermatology, 134, 11, 2709-2718 The Journal of investigative dermatology, 134, 11, 2709-2718 The Journal of investigative dermatology, 134, 11, 2709-2718 2014/11 Refereed Disclose to all
Nakajima S, Kitoh A, Egawa G, Natsuaki Y, Nakamizo S, Moniaga CS, Otsuka A, Honda T, Hanakawa S, Amano W, Iwakura Y, Nakae S, Kubo M, Miyachi Y, Kabashima K Nakajima S, Kitoh A, Egawa G, Natsuaki Y, Nakamizo S, Moniaga CS, Otsuka A, Honda T, Hanakawa S, Amano W, Iwakura Y, Nakae S, Kubo M, Miyachi Y, Kabashima K Nakajima S, Kitoh A, Egawa G, Natsuaki Y, Nakamizo S, Moniaga CS, Otsuka A, Honda T, Hanakawa S, Amano W, Iwakura Y, Nakae S, Kubo M, Miyachi Y, Kabashima K IL-17A as an inducer for Th2 immune responses in murine atopic dermatitis models. IL-17A as an inducer for Th2 immune responses in murine atopic dermatitis models. IL-17A as an inducer for Th2 immune responses in murine atopic dermatitis models. The Journal of investigative dermatology, 134, 8, 2122-2130 The Journal of investigative dermatology, 134, 8, 2122-2130 The Journal of investigative dermatology, 134, 8, 2122-2130 2014/08 Refereed Disclose to all
Nakashima C, Otsuka A, Kitoh A, Honda T, Egawa G, Nakajima S, Nakamizo S, Arita M, Kubo M, Miyachi Y, Kabashima K Nakashima C, Otsuka A, Kitoh A, Honda T, Egawa G, Nakajima S, Nakamizo S, Arita M, Kubo M, Miyachi Y, Kabashima K Nakashima C, Otsuka A, Kitoh A, Honda T, Egawa G, Nakajima S, Nakamizo S, Arita M, Kubo M, Miyachi Y, Kabashima K Basophils regulate the recruitment of eosinophils in a murine model of irritant contact dermatitis. Basophils regulate the recruitment of eosinophils in a murine model of irritant contact dermatitis. Basophils regulate the recruitment of eosinophils in a murine model of irritant contact dermatitis. The Journal of allergy and clinical immunology, 134, 1, 100-107 The Journal of allergy and clinical immunology, 134, 1, 100-107 The Journal of allergy and clinical immunology, 134, 1, 100-107 2014/07 Refereed Disclose to all
Otsuka A, Doi H, Egawa G, Maekawa A, Fujita T, Nakamizo S, Nakashima C, Nakajima S, Watanabe T, Miyachi Y, Narumiya S, Kabashima K Otsuka A, Doi H, Egawa G, Maekawa A, Fujita T, Nakamizo S, Nakashima C, Nakajima S, Watanabe T, Miyachi Y, Narumiya S, Kabashima K Otsuka A, Doi H, Egawa G, Maekawa A, Fujita T, Nakamizo S, Nakashima C, Nakajima S, Watanabe T, Miyachi Y, Narumiya S, Kabashima K Possible new therapeutic strategy to regulate atopic dermatitis through upregulating filaggrin expression. Possible new therapeutic strategy to regulate atopic dermatitis through upregulating filaggrin expression. Possible new therapeutic strategy to regulate atopic dermatitis through upregulating filaggrin expression. The Journal of allergy and clinical immunology, 133, 1, 139-146 The Journal of allergy and clinical immunology, 133, 1, 139-146 The Journal of allergy and clinical immunology, 133, 1, 139-146 2014/01 Refereed Disclose to all
Ono S, Nakajima S, Otsuka A, Miyachi Y, Kabashima K Ono S, Nakajima S, Otsuka A, Miyachi Y, Kabashima K Ono S, Nakajima S, Otsuka A, Miyachi Y, Kabashima K Pigmented purpuric dermatitis with high expression levels of serum TARC/CCL17 and epidermal TSLP. Pigmented purpuric dermatitis with high expression levels of serum TARC/CCL17 and epidermal TSLP. Pigmented purpuric dermatitis with high expression levels of serum TARC/CCL17 and epidermal TSLP. European journal of dermatology : EJD, 23, 5, 701-702 European journal of dermatology : EJD, 23, 5, 701-702 European journal of dermatology : EJD, 23, 5, 701-702 2013/09 Refereed Disclose to all
Mori T, Kabashima K, Fukamachi S, Kuroda E, Sakabe J, Kobayashi M, Nakajima S, Nakano K, Tanaka Y, Matsushita S, Nakamura M, Tokura Y Mori T, Kabashima K, Fukamachi S, Kuroda E, Sakabe J, Kobayashi M, Nakajima S, Nakano K, Tanaka Y, Matsushita S, Nakamura M, Tokura Y Mori T, Kabashima K, Fukamachi S, Kuroda E, Sakabe J, Kobayashi M, Nakajima S, Nakano K, Tanaka Y, Matsushita S, Nakamura M, Tokura Y D1-like dopamine receptors antagonist inhibits cutaneous immune reactions mediated by Th2 and mast cells. D1-like dopamine receptors antagonist inhibits cutaneous immune reactions mediated by Th2 and mast cells. D1-like dopamine receptors antagonist inhibits cutaneous immune reactions mediated by Th2 and mast cells. Journal of dermatological science, 71, 1, 37-44 Journal of dermatological science, 71, 1, 37-44 Journal of dermatological science, 71, 1, 37-44 2013/07 Refereed Disclose to all
Moniaga CS, Jeong SK, Egawa G, Nakajima S, Hara-Chikuma M, Jeon JE, Lee SH, Hibino T, Miyachi Y, Kabashima K Moniaga CS, Jeong SK, Egawa G, Nakajima S, Hara-Chikuma M, Jeon JE, Lee SH, Hibino T, Miyachi Y, Kabashima K Moniaga CS, Jeong SK, Egawa G, Nakajima S, Hara-Chikuma M, Jeon JE, Lee SH, Hibino T, Miyachi Y, Kabashima K Protease activity enhances production of thymic stromal lymphopoietin and basophil accumulation in flaky tail mice. Protease activity enhances production of thymic stromal lymphopoietin and basophil accumulation in flaky tail mice. Protease activity enhances production of thymic stromal lymphopoietin and basophil accumulation in flaky tail mice. The American journal of pathology, 182, 3, 841-851 The American journal of pathology, 182, 3, 841-851 The American journal of pathology, 182, 3, 841-851 2013/03 Refereed Disclose to all
Shiraishi N, Nomura T, Tanizaki H, Nakajima S, Narumiya S, Miyachi Y, Tokura Y, Kabashima K Shiraishi N, Nomura T, Tanizaki H, Nakajima S, Narumiya S, Miyachi Y, Tokura Y, Kabashima K Shiraishi N, Nomura T, Tanizaki H, Nakajima S, Narumiya S, Miyachi Y, Tokura Y, Kabashima K Prostaglandin E2-EP3 axis in fine-tuning excessive skin inflammation by restricting dendritic cell functions. Prostaglandin E2-EP3 axis in fine-tuning excessive skin inflammation by restricting dendritic cell functions. Prostaglandin E2-EP3 axis in fine-tuning excessive skin inflammation by restricting dendritic cell functions. PloS one, 8, 7 PloS one, 8, 7 PloS one, 8, 7 2013 Refereed Disclose to all
Otsuka A, Nakajima S, Kubo M, Egawa G, Honda T, Kitoh A, Nomura T, Hanakawa S, Sagita Moniaga C, Kim B, Matsuoka S, Watanabe T, Miyachi Y, Kabashima K Otsuka A, Nakajima S, Kubo M, Egawa G, Honda T, Kitoh A, Nomura T, Hanakawa S, Sagita Moniaga C, Kim B, Matsuoka S, Watanabe T, Miyachi Y, Kabashima K Otsuka A, Nakajima S, Kubo M, Egawa G, Honda T, Kitoh A, Nomura T, Hanakawa S, Sagita Moniaga C, Kim B, Matsuoka S, Watanabe T, Miyachi Y, Kabashima K Basophils are required for the induction of Th2 immunity to haptens and peptide antigens. Basophils are required for the induction of Th2 immunity to haptens and peptide antigens. Basophils are required for the induction of Th2 immunity to haptens and peptide antigens. Nature communications, 4 Nature communications, 4 Nature communications, 4 2013 Refereed Disclose to all
Nakajima S, Igyártó BZ, Honda T, Egawa G, Otsuka A, Hara-Chikuma M, Watanabe N, Ziegler SF, Tomura M, Inaba K, Miyachi Y, Kaplan DH, Kabashima K Nakajima S, Igyártó BZ, Honda T, Egawa G, Otsuka A, Hara-Chikuma M, Watanabe N, Ziegler SF, Tomura M, Inaba K, Miyachi Y, Kaplan DH, Kabashima K Nakajima S, Igyártó BZ, Honda T, Egawa G, Otsuka A, Hara-Chikuma M, Watanabe N, Ziegler SF, Tomura M, Inaba K, Miyachi Y, Kaplan DH, Kabashima K Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling. Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling. Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling. The Journal of allergy and clinical immunology, 129, 4, 1048-1055 The Journal of allergy and clinical immunology, 129, 4, 1048-1055 The Journal of allergy and clinical immunology, 129, 4, 1048-1055 2012/04 Refereed Disclose to all
Nakajima S, Watanabe H, Tohyama M, Sugita K, Iijima M, Hashimoto K, Tokura Y, Nishimura Y, Doi H, Tanioka M, Miyachi Y, Kabashima K Nakajima S, Watanabe H, Tohyama M, Sugita K, Iijima M, Hashimoto K, Tokura Y, Nishimura Y, Doi H, Tanioka M, Miyachi Y, Kabashima K Nakajima S, Watanabe H, Tohyama M, Sugita K, Iijima M, Hashimoto K, Tokura Y, Nishimura Y, Doi H, Tanioka M, Miyachi Y, Kabashima K High-mobility group box 1 protein (HMGB1) as a novel diagnostic tool for toxic epidermal necrolysis and Stevens-Johnson syndrome. High-mobility group box 1 protein (HMGB1) as a novel diagnostic tool for toxic epidermal necrolysis and Stevens-Johnson syndrome. High-mobility group box 1 protein (HMGB1) as a novel diagnostic tool for toxic epidermal necrolysis and Stevens-Johnson syndrome. Archives of dermatology, 147, 9, 1110-1112 Archives of dermatology, 147, 9, 1110-1112 Archives of dermatology, 147, 9, 1110-1112 2011/09 Refereed Disclose to all
Otsuka A, Kubo M, Honda T, Egawa G, Nakajima S, Tanizaki H, Kim B, Matsuoka S, Watanabe T, Nakae S, Miyachi Y, Kabashima K Otsuka A, Kubo M, Honda T, Egawa G, Nakajima S, Tanizaki H, Kim B, Matsuoka S, Watanabe T, Nakae S, Miyachi Y, Kabashima K Otsuka A, Kubo M, Honda T, Egawa G, Nakajima S, Tanizaki H, Kim B, Matsuoka S, Watanabe T, Nakae S, Miyachi Y, Kabashima K Requirement of interaction between mast cells and skin dendritic cells to establish contact hypersensitivity. Requirement of interaction between mast cells and skin dendritic cells to establish contact hypersensitivity. Requirement of interaction between mast cells and skin dendritic cells to establish contact hypersensitivity. PloS one, 6, 9 PloS one, 6, 9 PloS one, 6, 9 2011 Refereed Disclose to all
Tanizaki H, Egawa G, Inaba K, Honda T, Nakajima S, Moniaga CS, Otsuka A, Ishizaki T, Tomura M, Watanabe T, Miyachi Y, Narumiya S, Okada T, Kabashima K Tanizaki H, Egawa G, Inaba K, Honda T, Nakajima S, Moniaga CS, Otsuka A, Ishizaki T, Tomura M, Watanabe T, Miyachi Y, Narumiya S, Okada T, Kabashima K Tanizaki H, Egawa G, Inaba K, Honda T, Nakajima S, Moniaga CS, Otsuka A, Ishizaki T, Tomura M, Watanabe T, Miyachi Y, Narumiya S, Okada T, Kabashima K Rho-mDia1 pathway is required for adhesion, migration, and T-cell stimulation in dendritic cells. Rho-mDia1 pathway is required for adhesion, migration, and T-cell stimulation in dendritic cells. Rho-mDia1 pathway is required for adhesion, migration, and T-cell stimulation in dendritic cells. Blood, 116, 26, 5875-5884 Blood, 116, 26, 5875-5884 Blood, 116, 26, 5875-5884 2010/12 Refereed Disclose to all
Sugita K, Tohyama M, Watanabe H, Otsuka A, Nakajima S, Iijima M, Hashimoto K, Tokura Y, Miyachi Y, Kabashima K Sugita K, Tohyama M, Watanabe H, Otsuka A, Nakajima S, Iijima M, Hashimoto K, Tokura Y, Miyachi Y, Kabashima K Sugita K, Tohyama M, Watanabe H, Otsuka A, Nakajima S, Iijima M, Hashimoto K, Tokura Y, Miyachi Y, Kabashima K Fluctuation of blood and skin plasmacytoid dendritic cells in drug-induced hypersensitivity syndrome. Fluctuation of blood and skin plasmacytoid dendritic cells in drug-induced hypersensitivity syndrome. Fluctuation of blood and skin plasmacytoid dendritic cells in drug-induced hypersensitivity syndrome. The Journal of allergy and clinical immunology, 126, 2, 408-410 The Journal of allergy and clinical immunology, 126, 2, 408-410 The Journal of allergy and clinical immunology, 126, 2, 408-410 2010/08 Refereed Disclose to all
Nakajima S, Ohshima K, Kyogoku M, Miyachi Y, Kabashima K Nakajima S, Ohshima K, Kyogoku M, Miyachi Y, Kabashima K Nakajima S, Ohshima K, Kyogoku M, Miyachi Y, Kabashima K A case of intravascular large B-cell lymphoma with atypical clinical manifestations and analysis of CXCL12 and CXCR4 expression. A case of intravascular large B-cell lymphoma with atypical clinical manifestations and analysis of CXCL12 and CXCR4 expression. A case of intravascular large B-cell lymphoma with atypical clinical manifestations and analysis of CXCL12 and CXCR4 expression. Archives of dermatology, 146, 6, 686-687 Archives of dermatology, 146, 6, 686-687 Archives of dermatology, 146, 6, 686-687 2010/06 Refereed Disclose to all
Nakajima S, Honda T, Sakata D, Egawa G, Tanizaki H, Otsuka A, Moniaga CS, Watanabe T, Miyachi Y, Narumiya S, Kabashima K Nakajima S, Honda T, Sakata D, Egawa G, Tanizaki H, Otsuka A, Moniaga CS, Watanabe T, Miyachi Y, Narumiya S, Kabashima K Nakajima S, Honda T, Sakata D, Egawa G, Tanizaki H, Otsuka A, Moniaga CS, Watanabe T, Miyachi Y, Narumiya S, Kabashima K Prostaglandin I2-IP signaling promotes Th1 differentiation in a mouse model of contact hypersensitivity. Prostaglandin I2-IP signaling promotes Th1 differentiation in a mouse model of contact hypersensitivity. Prostaglandin I2-IP signaling promotes Th1 differentiation in a mouse model of contact hypersensitivity. Journal of immunology (Baltimore, Md. : 1950), 184, 10, 5595-5603 Journal of immunology (Baltimore, Md. : 1950), 184, 10, 5595-5603 Journal of immunology (Baltimore, Md. : 1950), 184, 10, 5595-5603 2010/05 Refereed Disclose to all
Moniaga CS, Egawa G, Kawasaki H, Hara-Chikuma M, Honda T, Tanizaki H, Nakajima S, Otsuka A, Matsuoka H, Kubo A, Sakabe J, Tokura Y, Miyachi Y, Amagai M, Kabashima K Moniaga CS, Egawa G, Kawasaki H, Hara-Chikuma M, Honda T, Tanizaki H, Nakajima S, Otsuka A, Matsuoka H, Kubo A, Sakabe J, Tokura Y, Miyachi Y, Amagai M, Kabashima K Moniaga CS, Egawa G, Kawasaki H, Hara-Chikuma M, Honda T, Tanizaki H, Nakajima S, Otsuka A, Matsuoka H, Kubo A, Sakabe J, Tokura Y, Miyachi Y, Amagai M, Kabashima K Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract. Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract. Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract. The American journal of pathology, 176, 5, 2385-2393 The American journal of pathology, 176, 5, 2385-2393 The American journal of pathology, 176, 5, 2385-2393 2010/05 Refereed Disclose to all
Honda T, Nakajima S, Egawa G, Ogasawara K, Malissen B, Miyachi Y, Kabashima K Honda T, Nakajima S, Egawa G, Ogasawara K, Malissen B, Miyachi Y, Kabashima K Honda T, Nakajima S, Egawa G, Ogasawara K, Malissen B, Miyachi Y, Kabashima K Compensatory role of Langerhans cells and langerin-positive dermal dendritic cells in the sensitization phase of murine contact hypersensitivity. Compensatory role of Langerhans cells and langerin-positive dermal dendritic cells in the sensitization phase of murine contact hypersensitivity. Compensatory role of Langerhans cells and langerin-positive dermal dendritic cells in the sensitization phase of murine contact hypersensitivity. The Journal of allergy and clinical immunology, 125, 5, 1154-1156 The Journal of allergy and clinical immunology, 125, 5, 1154-1156 The Journal of allergy and clinical immunology, 125, 5, 1154-1156 2010/05 Refereed Disclose to all

  • <<
  • >>
Title language:
History of staying abroad
Organization Department Research theme Country Period
National Institute of Allergic and Infectious Diseases Laboratory of Parasitic Diseases/Mucosal Immunity Section Skin commensals and host immunity United States of America 2015/04/01-2017/03/31